Impact Factor
4.67
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
19.85
2,394 total citations
h5-index
67
h5 depth signal
Cited share
96.1%
122 of 127
Total citations
2,394
Citation footprint
Metric signal
21.61
External metric anchor
Self-citation
1.1%
26 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
China
261
United States
119
Germany
41
Sweden
29
United Kingdom
29
Citing Fields of Research
Neurodegenerative Disorders
480
Neurosciences
64
Mitochondria
28
Noncoding RNA
26
Cell Biology
26
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
640
Articles tracked across the journal lifetime
Indexed publication set
127
122 cited publications
Last 12 months
73
6.1 publications/month
Open access share
100.0%
73 OA publications in the last year
Leading citing country
China
261 citing publications
Leading citing field
Neurodegenerative Disorders
480 citing publications
Latest tracked publication
19 October 2026
10 recent records available
Coverage & Indexing History
v9, 2020-
v1, 2012-
Active Coverage
Research Focus & Classification
Indexed Subjects
Neurodegenerative Disorders
Broad Categories
Neurology
Neurodegenerative Disorders
MeSH Descriptors
Nerve Degeneration
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
DOI 10.1186/s40035-026-00583-z
Open DOI
Exercise promotes lipophagy and mitigates cognitive impairment in Alzheimer’s disease through downregulating microglial TMEM9
Background: Autophagy-lysosomal dysfunction contributes to lipid droplet (LD) accumulation, which drives pathological progression in Alzheimer's disease (AD). Exercise is reported to restore autophagy-lysosomal function and attenuate cog...
02
DOI 10.1186/s40035-026-00593-x
Open DOI
ProSAAS overexpression arrests progression of motor deficits in a rat alpha-synuclein model of Parkinson’s disease
03
DOI 10.1186/s40035-026-00585-x
Open DOI
Levetiracetam restores GABAergic function and network homeostasis following Vps35 deficiency in excitatory neurons
Background: Alzheimer’s disease (AD) and related dementias (ADRDs) remain incurable, partly due to their complex and poorly understood pathogenic mechanisms. Loss of vacuolar protein sorting-associated protein 35 (VPS35), a core componen...
04
DOI 10.1186/s40035-026-00588-8
Open DOI
The glymphatic–lymphatic axis: known pathways, open questions, and emerging roles in neurodegeneration
The discovery of the glymphatic system and meningeal lymphatic vessels has transformed our understanding of brain fluid homeostasis and central–peripheral communication, highlighting the critical role of cerebrospinal fluid in brain clea...
05
DOI 10.1186/s40035-026-00590-0
Open DOI
Fyn kinase: a key mechanistic regulator and therapeutic target in tauopathy and neurodegenerative diseases
Synaptic dysfunction and intracellular accumulation of pathological tau are convergent hallmarks of Alzheimer’s disease (AD) and related tauopathies, yet the molecular mechanisms coupling these events to widespread neurodegeneration rema...
06
DOI 10.1186/s40035-026-00587-9
Open DOI
Microglia-derived extracellular vesicle content as a biomarker for early detection of Alzheimer’s disease
Alzheimer’s disease (AD) is a progressive neurodegenerative disorder characterized by amyloid-beta aggregation, tau pathology, synaptic dysfunction, and neuronal loss. While current biomarker frameworks such as the A/T/N classification h...
07
DOI 10.1186/s40035-026-00584-y
Open DOI
Large-scale genetic profiling of hereditary ataxias in China: implications for a stepwise molecular diagnostic strategy
Background: Hereditary ataxias are clinically and genetically heterogeneous, which complicates clinical management, genetic counseling, and scientific research. Efficient molecular diagnosis is essential but requires molecular epidemiolo...
08
09
DOI 10.1186/s40035-026-00586-w
Open DOI
Species-dependent activities of the PINK1–parkin axis
In vitro studies have established that PTEN-induced putative kinase 1 (PINK1) and parkin are central regulators of mitophagy, and loss-of-function mutations in either gene can cause early-onset Parkinson’s disease (PD). Although various ...
10
DOI 10.1186/s40035-026-00577-x
Open DOI
Brain region-resolved pharmacodynamics of an antisense oligonucleotide targeting human SNCA 3′UTR in BAC-hSNCA rats
Background: Alpha-synuclein accumulation contributes to Parkinson’s disease (PD), and lowering α-synuclein expression is a candidate disease-modifying approach. The brain-wide pharmacodynamic profile of human-targeting antisense oligonuc...
Bibliographical & Catalog Notes
Mode of access: World Wide Web.