Impact Factor
4.02
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
5.42
1,520 total citations
h5-index
50
h5 depth signal
Cited share
77.2%
230 of 298
Total citations
1,520
Citation footprint
Metric signal
4.93
External metric anchor
Self-citation
1.0%
15 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
United States
438
China
331
United Kingdom
295
Germany
167
Netherlands
126
Citing Fields of Research
Cancer & Development
335
Oncology
322
Immunology
238
Genetics & Genomics
128
Liver & Colon Cancer
128
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
10,320
Articles tracked across the journal lifetime
Indexed publication set
298
230 cited publications
Last 12 months
157
13.1 publications/month
Open access share
49.0%
77 OA publications in the last year
Leading citing country
United States
438 citing publications
Leading citing field
Cancer & Development
335 citing publications
Latest tracked publication
18 October 2026
10 recent records available
Coverage & Indexing History
v97n1, Jan. 1969-
v97n1, Jan. 1969-
Active Coverage
Research Focus & Classification
Indexed Subjects
Oncology
Cancer & Development
Immunology
Lung Cancer
Breast Cancer
Musculoskeletal & Nerve Cancer
Liver & Colon Cancer
Issuing & Sponsoring Bodies
Contributor
Pathological Society of Great Britain and Ireland
Broad Categories
Pathology
Cancer & Development
Musculoskeletal & Nerve Cancer
Digestive Disorders
Urology
Immunology
Cell Differentiation & Development
MeSH Descriptors
Pathology
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
DOI 10.1002/path.70144
Open DOI
Integrated molecular and histopathological analysis reveals genotype–phenotype correlations and diagnostic challenges in endometrial stromal sarcoma
Endometrial stromal sarcomas (ESSs) comprise a heterogeneous group of uterine mesenchymal tumors characterized by diverse morphological features and recurrent molecular alterations. Although molecular classification has substantially imp...
02
DOI 10.1002/path.70137
Open DOI
FOXC2 regulates macrophage polarization and osteosarcoma progression via the CXCL12 / CXCR4 signaling axis
Forkhead box protein C2 (FOXC2) is frequently overexpressed in osteosarcoma (OS) and has been implicated in tumor proliferation and metastasis; however, its role in regulating the tumor immune microenvironment remains poorly understood. ...
03
DOI 10.1002/path.70136
Open DOI
Molecular landscape of anaplastic transformation in pancreatic ductal adenocarcinoma
Pancreatic ductal adenocarcinoma (PDAC) with anaplastic features is a rare and highly aggressive subtype with an extremely poor prognosis. However, the molecular mechanisms underlying anaplastic transformation remain poorly understood. T...
04
DOI 10.1002/path.70127
Open DOI
Molecular, spatial, and AI ‐enabled technologies for predicting neoadjuvant therapy response in locally advanced rectal cancer
Pretreatment prediction of neoadjuvant response remains an unresolved challenge in locally advanced rectal cancer, where management is moving towards risk‐adapted and response‐oriented treatment strategies. Prediction remains difficult b...
05
DOI 10.1002/path.70134
Open DOI
From microbial tryptophan metabolism to crystal injury: an IPA – AhR checkpoint in calcium oxalate nephropathy
Calcium oxalate nephrolithiasis is commonly viewed as a consequence of urinary supersaturation, although crystal retention and renal injury are also influenced by gut microbiota dysbiosis and local tissue environment. In a recent study b...
06
DOI 10.1002/path.70135
Open DOI
Gut microbiota‐derived tryptophan metabolite attenuates calcium oxalate nephropathy by suppressing renal tubular cell apoptosis through the AhR – SOCS3 – STAT1 axis
Calcium oxalate (CaOx) nephropathy is a highly prevalent urological disease worldwide. Gut microbiota dysbiosis and host metabolic dysregulation are recognized as pivotal drivers in disease pathogenesis, yet the underlying mechanisms rem...
07
DOI 10.1002/path.70140
Open DOI
Patient iPSC ‐derived astrocytes reveal impaired homeostasis and reactive‐like features in propionic acidemia
Propionic acidemia (PA) is a neurometabolic disorder caused by propionyl‐CoA carboxylase deficiency with frequent neurological involvement, yet cell‐type‐specific mechanisms remain poorly defined. We established human induced pluripotent...
08
DOI 10.1002/path.70146
Open DOI
Issue Information
09
DOI 10.1002/path.70128
Open DOI
Rethinking colitis: MrgprD puts macrophages in the driver's seat †
There is increasing evidence to suggest that dysregulated macrophages play an important role in the promotion and maintenance of inflammatory bowel disease (IBD), yet the underlying mechanisms remain unclear. A recent study published by ...
10
DOI 10.1002/path.70125
Open DOI
MrgprD ‐mediated macrophage polarization drives chemically induced colitis in mice
Inflammatory bowel disease (IBD) is characterized by aberrant immune responses in the gut. Mas‐related G protein‐coupled receptor D (MrgprD), which is primarily expressed in sensory neurons of the dorsal root ganglia (DRG), is known to p...
Bibliographical & Catalog Notes
Assumes vol. numbering of the earlier title.
Also issued online.
Official journal of the Pathological Society of Great Britain and Ireland.
Vol. 157, no. 1 (JAN 1989)19900917 Notice not published Printed on acid-free paper