Impact Factor
3.47
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
3.30
614 total citations
h5-index
34
h5 depth signal
Cited share
66.8%
131 of 196
Total citations
614
Citation footprint
Metric signal
3.13
External metric anchor
Self-citation
5.4%
33 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
United States
692
Germany
75
United Kingdom
68
Canada
60
France
45
Citing Fields of Research
Physiology
785
Cell Biology
510
Cellular Membrane Transporters & Receptors
256
Cardiology
86
Musculoskeletal Disorders
55
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
9,448
Articles tracked across the journal lifetime
Indexed publication set
196
131 cited publications
Last 12 months
79
6.6 publications/month
Open access share
30.4%
24 OA publications in the last year
Leading citing country
United States
692 citing publications
Leading citing field
Physiology
785 citing publications
Latest tracked publication
19 October 2026
10 recent records available
Coverage & Indexing History
v48n6,Jul. 1965-
v1n1,Sep. 20, 1918-
Active Coverage
Active Coverage
Active Coverage
Research Focus & Classification
Indexed Subjects
Physiology
Cell Biology
Cellular Membrane Transporters & Receptors
Ophthalmology
Issuing & Sponsoring Bodies
Contributor
Rockefeller Institute for Medical Research
Contributor
Rockefeller Institute
Contributor
Society of General Physiologists
Broad Categories
Physiology
Cell Biology
Epithelial Barriers
Cardiology
MeSH Descriptors
Physiology
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
DOI 10.1085/jgp.202614130
Open DOI
Dissecting diastolic stiffness
JGP study (Shahid et al. https://doi.org/10.1085/jgp.202614028) reveals sex-based differences in the contributors to diastolic stress in cardiomyocytes from heart failure with preserved ejection fraction model mice.
02
DOI 10.1085/jgp.202614014
Open DOI
Early disruptions in brain capillary blood flow control in a mouse model of Alzheimer’s disease
In Alzheimer’s disease (AD), vascular dysfunction and impaired control of brain blood flow can disrupt cellular bioenergetics decades before cognitive decline. Identifying the earliest vascular changes is therefore critical for developin...
03
DOI 10.1085/jgp.20121084408242026c
Open DOI
Correction: Allosteric coupling in ligand-gated ion channels
04
DOI 10.1085/jgp.202613982
Open DOI
Chamber-specific regulation of myofilament Ca2+ sensitivity by nNOSβ via troponin I phosphorylation: Predominance in the right ventricle
Neuronal nitric oxide (NO) synthase (nNOS) is an important regulator of excitation–contraction coupling in cardiomyocytes, yet its chamber-specific role in right ventricular cardiomyocytes (RVCMs) remains incompletely understood. In this...
05
DOI 10.1085/jgp.202614024
Open DOI
State-dependent inhibition of LRRC8 volume-regulated anion channels by DCPIB
DCPIB is the prototypical inhibitor of leucine-rich repeat containing protein 8 volume-regulated anion channels (VRACs), yet the structural basis and state dependence of inhibition remain unclear. Here, we used chimeric channels, targete...
06
DOI 10.1085/jgp.202513840
Open DOI
A fluorescent non-hydrolyzable probe for the nucleotide-binding sites of KATP
Neuroendocrine ATP-sensitive K+ channels (KATP) comprise four pore-forming subunits (Kir6.2), each associated with a modulatory sulfonylurea receptor subunit (SUR1). ATP/ADP binding to Kir6.2 inhibits KATP; MgATP/MgADP binding to two dif...
07
DOI 10.1085/jgp.202614032
Open DOI
The proximal N terminus of IRAG is required for potentiation of HCN4 channels
The inositol triphosphate-associated, ER transmembrane proteins inositol triphosphate receptor-associated guanylate kinase substrate (IRAG) and lymphoid restricted membrane protein (LRMP) are isoform-specific regulators of the hyperpolar...
08
DOI 10.1085/jgp.202614031
Open DOI
The many faces of Orai1 dysfunction: Molecular mechanisms and clinical manifestations
Orai1, the pore-forming subunit of the calcium (Ca2+) release-activated Ca2+ (CRAC) channel, plays a central role in store-operated Ca2+ entry (SOCE) in animal cells and thereby serves as a key regulator of intracellular Ca2+ homeostasis...
09
DOI 10.1085/jgp.20251392308102026c
Open DOI
Correction: Evolution, hibernation, and inactivation of voltage-gated Na channels
10
DOI 10.1085/jgp.202614015
Open DOI
A hERG blocker facilitates K+ channel current by promoting pore opening while blocking
Many drugs that block voltage-gated K+ channels encoded by the human ether-à-go-go–related gene (hERG) can cause long QT syndrome and life-threatening cardiac arrhythmias, yet the molecular mechanisms that determine this risk remain uncl...
Bibliographical & Catalog Notes
Title from cover.
Supplements accompany some numbers.
Available also online.
Microfilm. v. 1 (1918)-30 (1947). 16 microfilm reels : negative ; 35 mm.
Official organ of the Society of General Physiologists, Sept. 1960-
Issued by the Rockefeller Institute for Medical Research, 1918-Mar. 1958; by the Rockefeller Institute, May 1958-
Vol. 90, no. 1 (1987)19921002 Notice not published Printed on acid-free paper