Impact Factor
4.75
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
11.19
4,267 total citations
h5-index
100
h5 depth signal
Cited share
83.1%
339 of 408
Total citations
4,267
Citation footprint
Metric signal
11.24
External metric anchor
Self-citation
1.6%
68 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
United States
1,521
Germany
299
China
265
United Kingdom
247
France
234
Citing Fields of Research
Immunology
1,698
Hematology
175
Neurodegenerative Disorders
146
Cancer & Development
146
Cancer, Autophagy & Apoptosis
141
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
26,025
Articles tracked across the journal lifetime
Indexed publication set
408
339 cited publications
Last 12 months
176
14.7 publications/month
Open access share
47.2%
83 OA publications in the last year
Leading citing country
United States
1,521 citing publications
Leading citing field
Immunology
1,698 citing publications
Latest tracked publication
20 October 2026
10 recent records available
Coverage & Indexing History
v122n3, Sept. 1965-
v1n1, Jan. 1, 1896-
Active Coverage
Active Coverage
Active Coverage
Research Focus & Classification
Indexed Subjects
Immunology
Issuing & Sponsoring Bodies
Contributor
Rockefeller Institute for Medical Research
Contributor
Rockefeller Institute
Contributor
Rockefeller University
Broad Categories
Allergy and Immunology
Medicine
Immunology
MeSH Descriptors
Medicine
Research
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
DOI 10.1084/jem.20260507
Open DOI
A genetically regulated SKAP2 switch modulates human T lymphocyte adhesion and migration
Genome-wide association studies investigating autoimmune disorders may reveal previously unrecognized regulators of immune cell functions. A Crohn’s disease risk allele was associated with increased expression in T lymphocytes of the ada...
02
DOI 10.1084/jem.20251898
Open DOI
Dysregulated AKT signaling reprograms osteosarcoma to drive selective reliance on EP300
Cancers depend on transcriptional control to maintain the malignant cell state. EP300 and CBP are paralogous master epigenetic enzymes that control normal and malignant transcription. Using a cancer-wide integrative chemical-genetic anal...
03
DOI 10.1084/jem.20251245
Open DOI
Cerebrospinal fluid and dural accumulation of α-synuclein impairs CNS clearance in Parkinson’s disease
Parkinson’s disease (PD) is characterized by the build-up of α-synuclein (α-syn) in the brain, where it drives neurodegeneration. Importantly, aggregated α-syn also accumulates in the cerebrospinal fluid (CSF) of PD patients, and while i...
04
DOI 10.1084/jem.20261476
Open DOI
Disarming FcR-dependent neutrophil activation in MPO-ANCA glomerulonephritis
Using spatial profiling, Nishide et al. (https://doi.org/10.1084/jem.20260506) identified activated neutrophils in kidneys in anti-myeloperoxidase (MPO)–associated vasculitis. They developed one-armed antibodies that block pathogenic ant...
05
DOI 10.1084/jem.20260506
Open DOI
Epitope-diverse autoantibodies induce neutrophil–epithelial niches driving kidney injury in ANCA-associated glomerulonephritis
Autoantibody-mediated inflammation drives tissue injury in autoimmune diseases, yet how pathogenic antibodies evoke local immune activation remains poorly understood. Here, we show that spatially organized neutrophil–epithelial niches co...
06
DOI 10.1084/jem.20260939
Open DOI
Macrophages as programmable integrators of disease tolerance across lifespan
Clinical outcome may depend as much on the host’s ability to preserve tissue function as on its ability to eliminate pathogens. The former reflects disease tolerance, with macrophages emerging as programmable integrators of multicellular...
07
DOI 10.1084/jem.20260378
Open DOI
Turning cold tumors hot: Strategies to enhance antitumor immunity
Immunologically cold tumors remain a major obstacle to effective cancer immunotherapy because they lack sufficient immune infiltration and fail to sustain productive antitumor immune responses. Although substantial progress has been made...
08
DOI 10.1084/jem.20260043
Open DOI
Exogenous nitric oxide as an anti-infective agent
Both before and after the discovery that the high-output isoform of nitric oxide (NO) synthase (iNOS; NOS2) contributes to control of many infectious diseases, investigators have been treating infections with exogenous NO or NO-related m...
09
DOI 10.1084/jem.20261370
Open DOI
Launching an independent research laboratory and scientific career: Tools for a rapidly changing landscape
Scientific independence alone does not create a sustainable research program. Drawing on the 2026 Irving Cancer Immunology Symposium, we offer practical strategies for navigating uncertainty and limited resources to build a coherent rese...
10
DOI 10.1084/jem.20252319
Open DOI
The selected few: Mechanisms of HIV-1 reservoir persistence
Combination antiretroviral therapy durably suppresses HIV-1 replication yet leaves untouched a small, highly specialized population of infected cells that sustains lifelong viral persistence and remains the principal obstacle to cure. In...
Bibliographical & Catalog Notes
Supplements accompany some issues.
Some issues have supplements which carry the numbering of the main title and also a separate supplement numbering, e.g. Vol. 34, no. 6 (Dec. 1, 1921) also called Supplement no. 1.
Also issued online.
Microfilm. v.1 (1896)-v.116 (1962),v.123 (1966)-v.132 (1970) + General Index v. 1 (1896)-v.60 (1934). 137 microfilm reels : negative ; 35 mm.
Issued by the Rockefeller Institute for Medical Research, Feb. 1905-Aug. 1958; by the Rockefeller Institute, Sept. 1958- ; by Rockefeller University, <July 1987->
Indexes: Vols. 1-20, 1896-1914. 1 v; Vols. 21-40, 1915-24. 1 v; Vols. 41-60, 1925-34. 1 v.
Vol. 180, no. 3 (SEPT 1994)19950517 Notice not published Printed on acid-free paper
digitized 2023