Impact Factor
3.80
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
5.37
289 total citations
h5-index
27
h5 depth signal
Cited share
95.0%
57 of 60
Total citations
289
Citation footprint
Metric signal
5.03
External metric anchor
Self-citation
2.8%
8 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
United States
185
Canada
50
France
45
China
35
United Kingdom
27
Citing Fields of Research
Musculoskeletal Disorders
316
Physiology
58
Cell Biology
53
Cell Differentiation & Development
44
Stem Cell Biology
43
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
542
Articles tracked across the journal lifetime
Indexed publication set
60
57 cited publications
Last 12 months
55
4.6 publications/month
Open access share
100.0%
55 OA publications in the last year
Leading citing country
United States
185 citing publications
Leading citing field
Musculoskeletal Disorders
316 citing publications
Latest tracked publication
12 October 2026
10 recent records available
Coverage & Indexing History
v1, 2011-
Active Coverage
Research Focus & Classification
Indexed Subjects
Musculoskeletal Disorders
Broad Categories
Physiology
Musculoskeletal Disorders
MeSH Descriptors
Muscle, Skeletal
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
DOI 10.1186/s13395-026-00452-3
Open DOI
Nonsense readthrough by ataluren prevents physical functional decline in aged dysferlinopathy mouse model
02
DOI 10.1186/s13395-026-00454-1
Open DOI
MyHC–SERCA mismatch as a marker of fiber-type remodeling in aging human skeletal muscle
03
DOI 10.1186/s13395-026-00455-0
Open DOI
Profibrotic activation of fibro-adipogenic progenitors impairs myogenesis through extracellular matrix synthesis and soluble factors
Background: The primary source of fibrosis in skeletal muscle is fibro-adipogenic progenitors (FAPs) which deposit extensive extracellular matrix (ECM). FAPs are key in supporting regeneration and myogenesis in healthy muscle but become ...
04
DOI 10.1186/s13395-026-00453-2
Open DOI
Klhl41b orchestrates sarcomere assembly and growth to drive skeletal muscle hypertrophy in vivo
Nemaline myopathy (NM) is a clinically and genetically heterogeneous disease caused by mutations in sarcomere-associated genes, with congenital forms repre
05
06
DOI 10.1186/s13395-026-00451-4
Open DOI
Galectin-1 therapy recalibrates lipid remodeling in skeletal muscle and enhances muscle function in a model of LGMD2B/R2
07
08
DOI 10.1186/s13395-026-00449-y
Open DOI
fishROI: a specialized workflow for semi-automated muscle morphometry analysis in teleosts
Background: Quantitative histological analysis of skeletal muscle morphometry provides critical insights into muscle physiology but remains labor-intensive and technically demanding. While recent developments in machine learning-based im...
09
DOI 10.1186/s13395-026-00450-5
Open DOI
Assessment of emerin gene transfer in models of X-linked Emery-Dreifuss muscular dystrophy
10
DOI 10.1186/s13395-026-00446-1
Open DOI