Impact Factor
4.10
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
4.56
3,495 total citations
h5-index
43
h5 depth signal
Cited share
86.8%
716 of 825
Total citations
3,495
Citation footprint
Metric signal
4.80
External metric anchor
Self-citation
6.3%
221 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
China
2,062
United States
177
Turkey
125
Japan
83
South Korea
46
Citing Fields of Research
Kidney Diseases
2,371
Urology
779
Diabetes
233
Cardiology
225
Transplantation
191
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
6,617
Articles tracked across the journal lifetime
Indexed publication set
825
716 cited publications
Last 12 months
503
41.9 publications/month
Open access share
100.0%
503 OA publications in the last year
Leading citing country
China
2,062 citing publications
Leading citing field
Kidney Diseases
2,371 citing publications
Latest tracked publication
15 October 2026
10 recent records available
Coverage & Indexing History
v10n1, 1987-
v10n1, 1987-
Active Coverage
Active Coverage
Research Focus & Classification
Indexed Subjects
Kidney Diseases
Broad Categories
Nephrology
Urology
MeSH Descriptors
Acute Kidney Injury
Uremia
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
02
DOI 10.1080/0886022x.2026.2728524
Open DOI
External validation and comparative performance of four risk scores for acute kidney injury in critically ill adults
03
DOI 10.1080/0886022x.2026.2745291
Open DOI
Acute kidney disease after acute kidney injury: incidence, risk factors, and association with de novo chronic kidney disease
04
05
DOI 10.1080/0886022x.2026.2729639
Open DOI
Genome-wide CRISPR screens identify shared and context-specific regulators of podocyte resistance to oxidative injury
Podocyte injury is a convergent driver of glomerular disease progression, yet the genetic determinants that govern podocyte vulnerability to oxidative stress remain incompletely defined. Here, we performed genome-wide CRISPR-Cas9 knockou...
06
DOI 10.1080/0886022x.2026.2741770
Open DOI
Progress on the related mechanisms of vascular calcification in autogenous arteriovenous fistula in hemodialysis patients
07
08
DOI 10.1080/0886022x.2026.2738248
Open DOI
Biological age acceleration and cardiovascular–kidney–metabolic syndrome progression: evidence from UK Biobank and NHANES
09
DOI 10.1080/0886022x.2026.2736897
Open DOI
A bibliometric analysis of trace elements and diabetic kidney disease: scientific hotspots and research evolution
10
DOI 10.1080/0886022x.2026.2738974
Open DOI
Association of environmental xylene exposure with diabetic kidney disease: a cross-sectional analysis and multi-omics evaluation of immune microenvironment mechanisms
Diabetic kidney disease (DKD) progression is driven by residual inflammation, potentially exacerbated by volatile organic compounds (VOCs). However, the impact of xylene exposure on the DKD immune microenvironment remains unclear. We eva...
Bibliographical & Catalog Notes
Imprint varies.
Print issues ceased with v. 33, no. 10 (2011).
Also issued online.
(1989)19890915 Notice not published Printed on acid-free paper