Impact Factor
3.76
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
3.66
955 total citations
h5-index
40
h5 depth signal
Cited share
77.9%
212 of 272
Total citations
955
Citation footprint
Metric signal
5.26
External metric anchor
Self-citation
2.3%
22 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
Germany
424
United States
226
Japan
134
China
98
Italy
77
Citing Fields of Research
Physiology
657
Cell Biology
243
Cellular Membrane Transporters & Receptors
200
Cardiology
162
Kidney Diseases
137
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
24,945
Articles tracked across the journal lifetime
Indexed publication set
272
212 cited publications
Last 12 months
91
7.6 publications/month
Open access share
60.4%
55 OA publications in the last year
Leading citing country
Germany
424 citing publications
Leading citing field
Physiology
657 citing publications
Latest tracked publication
20 October 2026
10 recent records available
Coverage & Indexing History
v302n1, 1968-
v302n1, 1968-
Active Coverage
Research Focus & Classification
Indexed Subjects
Physiology
Cell Biology
Kidney Diseases
Autonomic Function
Cardiology
Diabetes
Broad Categories
Physiology
Neurosciences
Autonomic Function
Urology
Cellular Membrane Transporters & Receptors
Cardiology
Musculoskeletal Disorders
MeSH Descriptors
Physiology, Comparative
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
DOI 10.1007/s00424-026-03225-5
Open DOI
ASIC1a as an integrating node in ischemic neurotoxicity: crosstalk with NMDAR and TRPM signaling
02
DOI 10.1007/s00424-026-03223-7
Open DOI
Glutamate tone in preBötzinger complex rhythmogenesis: ambient extracellular glutamate as a candidate excitability bias
The preBötzinger complex (preBötC) is essential for inspiratory rhythm generation, yet the biological processes that repeatedly establish the excitability state required for neuronal recruitment remain incompletely understood. This unres...
03
04
DOI 10.1007/s00424-026-03224-6
Open DOI
Revisiting the muscle spindle in ALS: Implications for spinal motor network dysfunction
Amyotrophic lateral sclerosis (ALS) is typically characterized by progressive degeneration of upper and lower motor neurons. However, the contribution of afferent sensory pathways to spinal motor network disorganization has received litt...
05
DOI 10.1007/s00424-026-03218-4
Open DOI
Cyclophilin D deficiency exacerbates diet-induced obesity and selectively enhances skeletal muscle mitochondrial respiration in male mice
Mitochondrial dysfunction is a hallmark of obesity-associated metabolic disease, with the mitochondrial permeability transition pore serving as a key regulator of cellular energy homeostasis. Cyclophilin D, encoded by Ppif , is the princ...
06
DOI 10.1007/s00424-026-03221-9
Open DOI
Differential alternative splicing in skeletal muscle after 14 days immobilization-induced atrophy in young men
Skeletal muscle disuse during injury decreases muscle mass and function. Previous studies have determined differential gene expression associated with atrophy, but changes in alternative splicing (AS) in response to unloading is poorly d...
07
DOI 10.1007/s00424-026-03219-3
Open DOI
Estimating the Starling equation parameters in living humans by using volume kinetic analysis
The transcapillary filtration of fluid is governed by the Starling forces. The purpose of the present study was to simultaneously estimate all Starling parameters in living humans by using likelihood mathematics. First, the capillary fil...
08
09
DOI 10.1007/s00424-026-03220-w
Open DOI
Sex-specific skeletal muscle fiber atrophy and reduced macrophage inflammation in a rodent transverse aortic constriction model
Heart failure is associated with systemic skeletal muscle remodeling, where sex-specific differences suggest differential peripheral involvement in disease progression. Skeletal muscle macrophages regulate angiogenesis and tissue remodel...
10
DOI 10.1007/s00424-026-03217-5
Open DOI
Acid-sensing ion channels in ischemic stroke: amplifying and constraining neuronal injury in a phase-specific framework
Summary: For decades, ischemic neuronal death was understood largely as a story about glutamate. Acid-sensing ion channels (ASICs) have revised that account. When cerebral blood flow stops, the penumbra can develop prolonged extracellula...
Bibliographical & Catalog Notes
Suppls. accompany some volumes.
Also issued online.
Text in English, French, and German, 1968- ; in English, <May 1997- >
(1988)19890821 Notice published Printed on acid-free paper