Impact Factor
4.59
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
29.27
6,659 total citations
h5-index
127
h5 depth signal
Cited share
64.3%
155 of 241
Total citations
6,659
Citation footprint
Metric signal
29.34
External metric anchor
Self-citation
0.6%
37 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
United States
442
United Kingdom
112
Australia
59
Germany
49
Canada
41
Citing Fields of Research
Oncology
512
Cancer & Development
359
Immunology
301
Cancer, Autophagy & Apoptosis
168
Genetics & Genomics
158
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
4,704
Articles tracked across the journal lifetime
Indexed publication set
241
155 cited publications
Last 12 months
102
8.5 publications/month
Open access share
2.0%
2 OA publications in the last year
Leading citing country
United States
442 citing publications
Leading citing field
Oncology
512 citing publications
Latest tracked publication
16 October 2026
10 recent records available
Coverage & Indexing History
v1n1, Oct. 2001-
v1n1, Oct. 2001-
Active Coverage
Research Focus & Classification
Indexed Subjects
Oncology
Immunology
Cancer & Development
Cancer, Autophagy & Apoptosis
Genetics & Genomics
Broad Categories
Neoplasms
Immunology
Cancer & Development
Cancer, Autophagy & Apoptosis
Genetics & Genomics
Lung Cancer
Cell Biology
MeSH Descriptors
Neoplasms
Research
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
DOI 10.1038/s41568-026-00996-y
Open DOI
Lean, mean, breast cancer-fighting machine
02
DOI 10.1038/s41568-026-00990-4
Open DOI
The urgent challenge of gastroesophageal cancer
03
DOI 10.1038/s41568-026-00985-1
Open DOI
Mapping the functional dark proteome in cancer
A generation after the genomics revolution, and with the advent of proteomic technologies, the dark proteome now stands as a potential new direction for cancer science and therapeutics. The dark proteome covers a vast terrain of previous...
04
DOI 10.1038/s41568-026-00984-2
Open DOI
Boosting CAR T cell efficacy in cancer with metabolic insights from design to diet
05
DOI 10.1038/s41568-026-00989-x
Open DOI
Supercentenarians’ superpower to suppress cancer
06
DOI 10.1038/s41568-026-00987-z
Open DOI
Engineered models of metastasis
07
DOI 10.1038/s41568-026-00986-0
Open DOI
Psychosocial burden in cancer survivors
08
DOI 10.1038/s41568-026-00980-6
Open DOI
From blunt-force treatment to precision: 25 years of transformation in cancer research
09
DOI 10.1038/s41568-026-00979-z
Open DOI
Replication stress in cancer: origins, consequences and therapeutic opportunities
Replication stress, defined as impaired DNA replication leading to genomic instability, is a central feature of tumour development and progression. In cancer cells, it arises from oncogene activation, transcription–replication conflicts,...
10
DOI 10.1038/s41568-026-00977-1
Open DOI
Viral mimicry escape as a necessary feature of malignant transformation
Malignant transformation is driven by disruption of pathways regulating proliferation and cell fate, but these same disruptions can create a collateral vulnerability: loss of transcriptional and epigenetic control over transposable eleme...
Bibliographical & Catalog Notes
Title from cover.
Also available online to subscribers.