Impact Factor
3.92
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
4.82
837 total citations
h5-index
40
h5 depth signal
Cited share
88.2%
157 of 178
Total citations
837
Citation footprint
Metric signal
4.81
External metric anchor
Self-citation
1.4%
12 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
China
286
United States
231
Japan
224
South Korea
210
Canada
148
Citing Fields of Research
Neurosciences
412
Cell Biology
315
Neurodegenerative Disorders
287
Physiology
118
Anesthetic & Pain Medicine
95
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
1,595
Articles tracked across the journal lifetime
Indexed publication set
178
157 cited publications
Last 12 months
106
8.8 publications/month
Open access share
100.0%
106 OA publications in the last year
Leading citing country
China
286 citing publications
Leading citing field
Neurosciences
412 citing publications
Latest tracked publication
17 October 2026
10 recent records available
Coverage & Indexing History
v1, 2008-
v1, 2008-
Active Coverage
Research Focus & Classification
Indexed Subjects
Neurodegenerative Disorders
Neurosciences
Cell Biology
Physiology
Issuing & Sponsoring Bodies
Contributor
BioMed Central Ltd
Broad Categories
Brain
Molecular Biology
Neurosciences
Neurodegenerative Disorders
Physiology
Anesthetic & Pain Medicine
Cancer & Development
MeSH Descriptors
Brain
Molecular Biology
Neurosciences
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
DOI 10.1186/s13041-026-01353-8
Open DOI
Comprehensive behavioral phenotyping of male Sulf1-deficient mice reveals reduced locomotor activity, altered forelimb gait, and reduced prepulse inhibition
Sulfatase 1 (Sulf1) is an extracellular sulfatase that regulates multiple signaling pathways by removing 6- O -sulfate from heparan sulfate. Although Sulf1 has established roles in development and cancer, its functions in the adult brain...
02
DOI 10.1186/s13041-026-01354-7
Open DOI
Organoids as advanced three-dimensional models for studying brain development, neurodegeneration, and translational therapeutics
Organoids are stem cell-derived three-dimensional (3D) multicellular constructs that recapitulate key cytoarchitectural, molecular, and functional properties of their tissue of origin. Two fundamentally distinct strategies govern their g...
03
DOI 10.1186/s13041-026-01352-9
Open DOI
PPM1H loss alters intrasynaptic alpha-synuclein localization in a LRRK2-dependent manner
Mutations in the LRRK2 gene are a major genetic cause of Parkinson’s disease (PD) and induce hyperphosphorylation of RAB GTPases through increased LRRK2 kinase activity. The phosphatase PPM1H counteracts this activity by selectively deph...
04
DOI 10.1186/s13041-026-01293-3
Open DOI
PPARγ–Wnt/β-catenin–HK2 axis associates with impaired neuroimmune homeostasis and metabolic dysregulation in MIA offspring
Maternal immune activation (MIA) during gestation elevates the risk for autism spectrum disorder (ASD), but the underlying neurobiological mechanisms require further elucidation. Using a lipopolysaccharide (LPS)-induced MIA rat model, of...
05
DOI 10.1186/s13041-026-01351-w
Open DOI
Impact of environmental enrichment and allostatic load induced by repeated mixing on the transcriptome of three porcine brain tissues
Environmental enrichment and mixing represent two management procedures in pig production with opposing effects: while environmental enrichment can promote welfare by reducing stress, repeated mixing often induces stress and can negative...
06
DOI 10.1186/s13041-026-01350-x
Open DOI
Spinal dehydroepiandrosterone sulfate induces convulsion-like behavior via NMDA receptor-mediated Ca2+ influx and sigma-1 receptor-mediated intracellular Ca2+ release
Dehydroepiandrosterone sulfate (DHEAS) is a neurosteroid that modulates spinal nociception and hyperalgesia. However, the immediate impact of an acute increase in spinal DHEAS remains poorly understood. Here, we report that intrathecal (...
07
DOI 10.1186/s13041-026-01345-8
Open DOI
Disease-specific dominant signalling pathways and signatures in cerebrovascular diseases
Cerebrovascular diseases (CVDs) comprise a heterogeneous group of brain diseases, including stroke, cerebral small vessel disease (CSVD), cerebral amyloid angiopathy (CAA), and vascular dementia (VaD), driven by distinct yet overlapping ...
08
DOI 10.1186/s13041-026-01348-5
Open DOI
Activation of alpha-2 adrenoceptors in locus coeruleus alleviates prenatal lipopolysaccharide-induced cognitive deficits in the offspring of rats
The locus coeruleus (LC), the brain’s principal source of norepinephrine, critically regulates cognitive function. While our previous work established that prenatal lipopolysaccharide (LPS) exposure causes cognitive deficits in rat offsp...
09
DOI 10.1186/s13041-026-01349-4
Open DOI
Knockdown of SENP7 attenuates cognitive impairment in offspring mice induced by maternal sevoflurane anesthesia partially through cGAS/STING/IRF3-mediated pyroptosis
Background: Exposure to sevoflurane (SEV) during pregnancy is associated with long-term cognitive impairment in offspring; however, the precise mechanisms remain unclear. This study aimed to investigate the role of Sentrin/SUMO-specific ...
10
DOI 10.1186/s13041-026-01346-7
Open DOI
Differential circadian time modulates of dentate gyrus and CA1 excitation-inhibition balance and seizure threshold
Circadian rhythms regulate seizure threshold and neuronal excitability in epilepsy. Temporal lobe seizures tend to arise more frequently during the diurnal phase relative to the nocturnal phase, yet dentate gyrus (DG) granule cells displ...
Bibliographical & Catalog Notes
Description based on: 1:1 (17 June 2008); title from caption (publisher's Web site, viewed Mar. 17, 2009).
Latest issue consulted: 2:7 (5 Mar. 2009) (viewed Mar. 17, 2009).
Articles added consecutively to current annual v.
Mode of access: World Wide Web.