Impact Factor
2.88
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
2.26
218 total citations
h5-index
17
h5 depth signal
Cited share
65.3%
66 of 101
Total citations
218
Citation footprint
Metric signal
1.78
External metric anchor
Self-citation
25.7%
56 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
Italy
226
China
74
Turkey
34
United States
32
Spain
26
Citing Fields of Research
Endocrinology & Metabolism
257
Diabetes
163
Endocrine Therapy
62
Human Reproduction
61
Adrenal Dysfunction
55
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
578
Articles tracked across the journal lifetime
Indexed publication set
101
66 cited publications
Last 12 months
61
6.1 publications/month
Open access share
6.6%
4 OA publications in the last year
Leading citing country
Italy
226 citing publications
Leading citing field
Endocrinology & Metabolism
257 citing publications
Latest tracked publication
1 September 2026
10 recent records available
Coverage & Indexing History
v46n1, 2021-
v46n1, 2021-
Research Focus & Classification
Indexed Subjects
Endocrinology & Metabolism
Diabetes
Human Reproduction
Endocrine Therapy
Broad Categories
Endocrinology
Endocrinology & Metabolism
Diabetes
Human Reproduction
Osteopathies
Adrenal Dysfunction
MeSH Descriptors
Endocrine System Diseases
Endocrinology
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
DOI 10.23736/s2724-6507.26.04628-2
Open DOI
Highlights of the July-September 2026 issue
02
DOI 10.23736/s2724-6507.26.04454-4
Open DOI
Cardiometabolic and renal complications of obesity: an updated comprehensive overview by SIO - Campania region
Obesity has reached epidemic proportions globally, with its prevalence nearly tripling since 1975. It is now recognized as a chronic, relapsing disease associated with increased morbidity and mortality due to its strong relationship with...
03
DOI 10.23736/s2724-6507.26.04558-6
Open DOI
Clinical effects of apolipoprotein C3 inhibitors on triglyceride levels and pancreatitis risk: a systematic review and meta-analysis
Novel therapies targeting apolipoprotein C3 (apoC3) have recently emerged as promising treatment options for hypertriglyceridemia and have been evaluated in phase II and III randomized clinical trials. This study aimed to perform an upda...
04
DOI 10.23736/s2724-6507.26.04593-8
Open DOI
Highlights of the April-June 2026 issue
05
DOI 10.23736/s2724-6507.26.04552-5
Open DOI
Uncomplicated diabetes and adverse in-hospital outcomes in tumor lysis syndrome: a national cohort analysis
06
DOI 10.23736/s2724-6507.26.04575-6
Open DOI
Obesity-cancer convergence in the destiny of an adipocyte
07
DOI 10.23736/s2724-6507.26.04541-0
Open DOI
Effects of testosterone undecanoate as add-on therapy in obese hypogonadal men that are late responders to tirzepatide: a pilot study
Article: Effects of testosterone undecanoate as add-on therapy in obese hypogonadal men that are late responders to tirzepatide: a pilot study - Minerva Endocrinol 2026 Mar 09 - Minerva Medica - Journals
08
DOI 10.23736/s2724-6507.25.04276-9
Open DOI
Dioxin, an endocrine disruptor, induces long term effects on DNA methylation in men after in-utero exposure
Prenatal exposure to dioxin, a known endocrine disruptor, after the Seveso accident of 1976 has been associated with thyroid dysfunction, metabolic syndrome and semen quality reduction. Experimental exposure to dioxin in utero produced e...
09
DOI 10.23736/s2724-6507.25.04322-2
Open DOI
Role of microRNA in type 1 diabetes pathogenesis and their therapeutic potential
Type 1 diabetes (T1D) is a chronic autoimmune disease characterized by the destruction of insulin-producing β-cells in the pancreatic islets, resulting in complete insulin deficiency. The pathogenesis of T1D is multifactorial and include...
10
DOI 10.23736/s2724-6507.25.04390-8
Open DOI
An overview of insulin resistance: from molecular mechanisms to medical interventions
Insulin resistance (IR) is a condition where insulin-targeted tissues (muscle, liver, and adipose tissue) fail to normally respond to insulin action, leading to impaired insulin signaling and hyperglycemia. It is common in individuals wi...
Bibliographical & Catalog Notes
Search interface in English and Italian.