Impact Factor
4.10
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
5.62
1,501 total citations
h5-index
37
h5 depth signal
Cited share
85.8%
236 of 275
Total citations
1,501
Citation footprint
Metric signal
8.23
External metric anchor
Self-citation
12.3%
184 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
China
320
United States
224
Brazil
125
Japan
121
Italy
70
Citing Fields of Research
Dentistry & Oral Medicine
1,028
Osteopathies
141
Immunology
102
Stem Cell Biology
62
Noncoding RNA
59
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
5,012
Articles tracked across the journal lifetime
Indexed publication set
275
236 cited publications
Last 12 months
137
11.4 publications/month
Open access share
74.5%
102 OA publications in the last year
Leading citing country
China
320 citing publications
Leading citing field
Dentistry & Oral Medicine
1,028 citing publications
Latest tracked publication
10 October 2026
10 recent records available
Coverage & Indexing History
v1, 1966-
v1, 1966-
v1, 1966-v35n1, Feb. 2000
Active Coverage
Research Focus & Classification
Indexed Subjects
Dentistry & Oral Medicine
Broad Categories
Dentistry
Dentistry & Oral Medicine
MeSH Descriptors
Periodontics
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
DOI 10.1111/jre.70183
Open DOI
Systemic RVX ‐208 Attenuates Experimental Periodontitis Through Inflammatory Host Modulation
Aims: This study investigated whether systemic treatment of RVX‐208, a selective BET protein inhibitor, could ameliorate experimental periodontitis in a prophylactic setting and examined transcriptomic changes associated with RVX‐208 in ...
02
03
DOI 10.1111/jre.70173
Open DOI
Periodontal Salutogenesis: Why Are Some People Resistant to Oral Microbial Dysbiosis and Destructive Inflammation?
Periodontitis research has traditionally focused on pathogenesis; however, understanding why some individuals remain resistant to the disease may be even more important. The concept of salutogenesis (“salutology”) has evolved around the ...
04
05
Aim: Peri‐implant tissues are more susceptible than gingiva to rapid inflammatory breakdown, but the cellular basis of this vulnerability remains unclear. This study investigated (1) whether clinically healthy peri‐implant mucosa already...
06
07
08
DOI 10.1111/jre.70167
Open DOI
The Systemic Impact of Peri‐Implantitis: A Systematic Review
Aim: To systematically evaluate the available evidence regarding the association between peri‐implantitis and systemic inflammatory biomarkers and/or systemic diseases, and to assess whether peri‐implantitis treatment influences systemic...
09
DOI 10.1111/jre.70163
Open DOI
Periodontitis, Edentulism, and Accelerated Biological Aging: A Population‐Based Study of Epigenetic Clocks
Aim: To examine whether periodontitis and its ultimate sequela (edentulism) are associated with accelerated epigenetic aging in older adults. Methods: A total of 1 926 adults aged ≥ 50 years with DNA‐methylation (DNAm) and oral health da...
10
DOI 10.1111/jre.70177
Open DOI
Periodontitis, Tooth Loss, and Risk of Cognitive Impairment and Dementia: A Systematic Review With Meta‐Analysis
Aim: To systematically review and meta‐analyze cohort studies evaluating associations of gingival inflammation, periodontitis, and tooth loss with cognitive decline and incident mild cognitive impairment or dementia. Methods: PROSPERO‐re...
Bibliographical & Catalog Notes
Also issued online.
Vol. 15, no. 1 (1980)19921022 Notice not published Printed on acid-free paper