Impact Factor
3.22
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
2.28
587 total citations
h5-index
30
h5 depth signal
Cited share
62.8%
164 of 261
Total citations
587
Citation footprint
Metric signal
2.17
External metric anchor
Self-citation
5.1%
30 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
United States
614
China
152
Japan
111
Canada
66
Germany
63
Citing Fields of Research
Neurodegenerative Disorders
532
Brain Neurological Disorders
305
Brain Imaging
286
Nervous System Disorders
187
Neuromuscular Disorders
119
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
11,966
Articles tracked across the journal lifetime
Indexed publication set
261
164 cited publications
Last 12 months
148
12.3 publications/month
Open access share
36.5%
54 OA publications in the last year
Leading citing country
United States
614 citing publications
Leading citing field
Neurodegenerative Disorders
532 citing publications
Latest tracked publication
20 October 2026
10 recent records available
Coverage & Indexing History
v24n4, Oct. 1965-
v24n4, Oct. 1965-
Active Coverage
Active Coverage
Research Focus & Classification
Indexed Subjects
Neurodegenerative Disorders
Brain Imaging
Brain Neurological Disorders
Neurosciences
Musculoskeletal Disorders
Neuromuscular Disorders
Issuing & Sponsoring Bodies
Contributor
American Association of Neuropathologists
Broad Categories
Neurology
Pathology
Neurodegenerative Disorders
Brain Imaging
Musculoskeletal Disorders
Cancer, Autophagy & Apoptosis
MeSH Descriptors
Nervous System Diseases
Neurology
Pathology
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
Human prion diseases, including sporadic Creutzfeldt-Jakob disease (sCJD) and Gerstmann-Sträussler-Scheinker disease (GSS), are fatal neurodegenerative disorders characterized by misfolded prion protein (PrP) accumulation. Although type ...
02
DOI 10.1093/jnen/nlag113
Open DOI
In This Issue
03
DOI 10.1093/jnen/nlag109
Open DOI
Pellagra encephalopathy in Huntington disease: An autopsy case
04
05
DOI 10.1093/jnen/nlag107
Open DOI
Autobiography series: Medievalia, music, and medicine
06
DOI 10.1093/jnen/nlag104
Open DOI
HOXD12 as an oncofetal protein expressed in WNT-activated medulloblastomas
Aberrant reactivation of embryonic patterning programs is a hallmark of cancer. We previously identified HOXD12 activation in aggressive oligodendrogliomas, but its broader significance across brain tumors remains unexamined. Here, we ev...
07
DOI 10.1093/jnen/nlag081
Open DOI
Cardiovascular risk factors and regional neuropathology in autopsy-confirmed Alzheimer disease
Cardiovascular risk factors are implicated in Alzheimer disease (AD) progression and its neuropathological hallmarks but their specific contributions to regional brain pathology within AD remain understudied. In this cohort study (n = 27...
08
DOI 10.1093/jnen/nlag106
Open DOI
In This Issue
09
DOI 10.1093/jnen/nlag086
Open DOI
CDKN2A/B status can be assessed by p16/MTAP immunohistochemistry as a practical surrogate in pleomorphic xanthoastrocytoma and high-grade astrocytoma with piloid features
The presence of a CDKN2A homozygous deletion (HD) plays an important role in the diagnostic approach of neuropathologists to several central nervous system (CNS) tumors. CDKN2A is located on chromosome 9p21, next to CDKN2B and MTAP, and ...
10
DOI 10.1093/jnen/nlag096
Open DOI
Epigenetic deregulation in adult gliomas: A key driver of molecular heterogeneity and precision oncology
Gliomas, particularly glioblastoma, are characterized by profound molecular and cellular heterogeneity that drives tumor progression, therapeutic resistance, and poor clinical outcomes. Increasing evidence indicates that epigenetic dereg...
Bibliographical & Catalog Notes
Also issued online.
Indexes: Vols. 1-15, 1942-56, in v. 16.
Vol. 46, no. 1 (JAN 1987)19890816 Notice published Printed on acid-free paper