Impact Factor
2.84
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
1.85
146 total citations
h5-index
17
h5 depth signal
Cited share
63.6%
56 of 88
Total citations
146
Citation footprint
Metric signal
1.83
External metric anchor
Self-citation
2.1%
3 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
United States
182
Australia
129
Brazil
111
Canada
97
United Kingdom
82
Citing Fields of Research
Obstetrics/Gynecology
357
Diabetes
139
Nutrition & Dietetics
122
Human Reproduction
88
Endocrinology & Metabolism
56
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
1,307
Articles tracked across the journal lifetime
Indexed publication set
88
56 cited publications
Last 12 months
58
4.8 publications/month
Open access share
56.9%
33 OA publications in the last year
Leading citing country
United States
182 citing publications
Leading citing field
Obstetrics/Gynecology
357 citing publications
Latest tracked publication
15 October 2026
10 recent records available
Coverage & Indexing History
v5n1, 2014-
v5n1, 2014- ; selected citations only before this date
Research Focus & Classification
Indexed Subjects
Obstetrics/Gynecology
Nutrition & Dietetics
Diabetes
Endocrinology & Metabolism
Inflammatory Bowel Diseases & Infections
Dairy & Animal Science
Issuing & Sponsoring Bodies
Contributor
International Society for Developmental Origins of Health and Disease
Broad Categories
Embryology
Medicine
Obstetrics/Gynecology
MeSH Descriptors
Developmental Biology
Disease Susceptibility
Prenatal Exposure Delayed Effects
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
DOI 10.1017/s2040174426100828
Open DOI
Neonatal handling modifies maternal care and lactational adaptations to gestational protein restriction
Prenatal nutritional conditions influence offspring metabolic health and may also program maternal physiology and caregiving during lactation. Early postnatal experience can modify maternal–infant interactions and thus modulate maternal ...
02
DOI 10.1017/s2040174426100889
Open DOI
Subclinical neonatal hypoxic stress: a hypothesis on transitional oxygen instability
The transition from fetal to neonatal life requires rapid adjustments in respiratory, cardiovascular, and autonomic regulation as the newborn establishes independent oxygenation. Although most infants achieve stable physiological adaptat...
03
DOI 10.1017/s2040174426100798
Open DOI
Mothers’ and partners’ changes in health behaviours during pregnancy: observations from the Growing Up in New Zealand Cohort
Improving modifiable health behaviours during pregnancy may benefit parental health and offspring outcomes. Although pregnancy may motivate lifestyle change in both mothers and partners, evidence on partner health behaviour change and wi...
04
DOI 10.1017/s204017442610083x
Open DOI
Maternal protein restriction during lactation programs peripheral insulin sensitivity and reduces UCP1 abundance in interscapular brown adipose tissue of adult male Wistar rats
Maternal protein restriction during lactation is associated with stunted growth and hyperphagia in adult offspring, yet its long-term effects on mitochondrial uncoupling-related proteins in interscapular brown adipose tissue (iBAT) remai...
05
DOI 10.1017/s2040174426100841
Open DOI
DOHaD ANZPac: bringing together the DOHaD community across Australia, New Zealand, and the Pacific
06
DOI 10.1017/s2040174426100713
Open DOI
Repositioning oral health within DOHaD: linking early childhood caries to life-course NCD prevention
The Developmental Origins of Health and Disease (DOHaD) paradigm has transformed our understanding of how early-life biological and social environments shape lifelong health trajectories. While DOHaD research across Oceania has focused p...
07
DOI 10.1017/s2040174426100865
Open DOI
Maternal obesity induces sex-specific gut microbiota changes associated with reduced fecal levels of endocannabinoid-like molecules in rat offspring at weaning
Maternal obesity alters the gut microbiota of offspring, although sex-specific effects remain poorly assessed before puberty. An interplay has emerged between the gut microbiota and fecal endocannabinoids, including oleoylethanolamide (O...
08
DOI 10.1017/s2040174426100804
Open DOI
Maternal exposure to fluorotelomer ethoxylates: magnetic resonance spectroscopy and imaging to study bioaccumulation during pregnancy and the long-term impact on offspring neuroanatomy
Fluorotelomer ethoxylates (FTEOs) are a new and unregulated class of per-/polyfluoroalkyl substances (PFAS) recognized to be persistent in the environment and known in the laboratory setting to have important health effects, such as caus...
09
DOI 10.1017/s2040174426100816
Open DOI
Growth faltering in infants: a contemporary review
Growth faltering, often referred to as failure to thrive (FTT), is a significant and frequently encountered issue in pediatric care. It denotes inadequate growth resulting from a lack of sufficient caloric intake, impaired nutrient absor...
10
DOI 10.1017/s2040174426100774
Open DOI
Maternal overnutrition during pregnancy: impacts on pancreatic proteome of the offspring
Maternal obesity, resulting from high caloric intake during pregnancy, can impair pancreatic development in the offspring and lead to an increased risk of diabetes. We hypothesized that maternal obesity during pregnancy results in reduce...
Bibliographical & Catalog Notes
Print issues ceased with v. 2 (2011).
Vol. 1, issue 1 preceded by a suppl. called Vol. 1, suppl.1 (Nov. 2009).
Also issued online.