Impact Factor
3.86
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
3.89
2,075 total citations
h5-index
57
h5 depth signal
Cited share
71.5%
403 of 564
Total citations
2,075
Citation footprint
Metric signal
3.56
External metric anchor
Self-citation
0.5%
11 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
China
2,926
United States
895
Iran
765
Italy
393
India
254
Citing Fields of Research
Noncoding RNA
1,346
Cell Biology
888
Cancer & Development
666
Cancer, Autophagy & Apoptosis
661
Stem Cell Biology
658
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
18,523
Articles tracked across the journal lifetime
Indexed publication set
564
403 cited publications
Last 12 months
141
11.8 publications/month
Open access share
35.5%
50 OA publications in the last year
Leading citing country
China
2,926 citing publications
Leading citing field
Noncoding RNA
1,346 citing publications
Latest tracked publication
18 October 2026
10 recent records available
Coverage & Indexing History
v67n1, Feb. 1966-
v67n1, Feb. 1966-
Active Coverage
Research Focus & Classification
Indexed Subjects
Cell Biology
Immunology
Stem Cell Biology
Physiology
Cancer, Autophagy & Apoptosis
Cancer & Development
Cell Differentiation & Development
Issuing & Sponsoring Bodies
Contributor
Wistar Institute of Anatomy and Biology
Broad Categories
Cell Biology
Physiology
Cancer, Autophagy & Apoptosis
Musculoskeletal Disorders
Cancer & Development
Noncoding RNA
Osteopathies
Stem Cell Biology
MeSH Descriptors
Cell Biology
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
DOI 10.1002/jcp.70235
Open DOI
Hepatic Macrophage STING Signaling in Liver Fibrosis: Mechanisms and Therapeutic Perspectives
Liver fibrosis (LF) arises from chronic liver injury, in which a persistently pro‐inflammatory microenvironment shaped by hepatic macrophages drives hepatic stellate cells (HSCs) activation and fibrosis progression. The cyclic GMP‐AMP sy...
02
DOI 10.1002/jcp.70237
Open DOI
Revisiting Cell Death in Colorectal Cancer: The Rise of Ferroptosis as a Therapeutic Opportunity
Colorectal cancer (CRC) remains a global health concern and a leading cause of cancer mortality, where therapeutic resistance persists as the central barrier to sustained response. Most conventional therapies rely on apoptosis, but CRC f...
03
DOI 10.1002/jcp.70239
Open DOI
Rev‐erb‐α in Skeletal Muscle Adaptation: Integrating Circadian Control Mechanisms and Exercise Dynamic Responses
Circadian rhythms orchestrate a host of biological processes in a time‐dependent manner to maintain homeostasis in response to exercise. Within this framework, Rev‐erb‐α, an intracellular nuclear receptor and transcriptional regulator, t...
04
DOI 10.1002/jcp.70241
Open DOI
Sestrin1 Ameliorates Senescence and Mediates the Protective Effects of Exercise Against Hepatic Abnormality in C57BL/6J Mice
Liver aging is characterized by structural remodeling and functional decline, frequently driven by mitochondrial dysfunction and senescence‐associated secretory phenotype (SASP). Sestrins (SESNs) are a family of stress‐inducible proteins...
05
DOI 10.1002/jcp.70238
Open DOI
SLC35F2 in Cancer Development and Progression
Solute carrier family 35 member F2 (SLC35F2) is a transporter located predominantly at the plasma membrane and Golgi apparatus and is predicted to contain ten transmembrane helices. Recent studies have identified SLC35F2 as the only curr...
06
Lipid oversupply induces insulin resistance in skeletal muscle (SkM), partly by altering mitochondrial function and dynamics. Although electrical pulse stimulation (EPS) improves insulin signaling in myotubes, the mechanisms contributing...
07
08
DOI 10.1002/jcp.70236
Open DOI
Issue Information
09
DOI 10.1002/jcp.70233
Open DOI
Molecular and Cellular Underpinnings of Primary Hypertension: An Integrative Mechanistic Framework
Hypertension is a highly prevalent clinical condition and a major contributor to global cardiovascular morbidity and mortality. Despite extensive research, its complex and multifaceted pathogenesis is yet to be fully elucidated. This pap...
10
Bibliographical & Catalog Notes
Also issued online.
(1989)19890816 Notice published Printed on acid-free paper