Impact Factor
4.67
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
11.61
2,723 total citations
h5-index
68
h5 depth signal
Cited share
88.0%
221 of 251
Total citations
2,723
Citation footprint
Metric signal
13.33
External metric anchor
Self-citation
1.2%
32 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
United States
602
Germany
80
France
79
United Kingdom
78
Australia
66
Citing Fields of Research
Immunology
890
Hematology
88
Allergies
82
Immunotherapy
53
Cancer, Autophagy & Apoptosis
43
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
4,679
Articles tracked across the journal lifetime
Indexed publication set
251
221 cited publications
Last 12 months
118
9.8 publications/month
Open access share
57.6%
68 OA publications in the last year
Leading citing country
United States
602 citing publications
Leading citing field
Immunology
890 citing publications
Latest tracked publication
19 October 2026
10 recent records available
Coverage & Indexing History
v33,Jan. 1977-
v33,Jan. 1977-
Active Coverage
Research Focus & Classification
Indexed Subjects
Immunology
Broad Categories
Allergy and Immunology
Transplantation
Immunology
MeSH Descriptors
Allergy and Immunology
Transplantation
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
DOI 10.1111/imr.70185
Open DOI
Hybrid Insulin Peptides in Type 1 Diabetes: How They Form and Why They Matter
Type 1 diabetes is a T cell‐mediated autoimmune disease that leads to the destruction of the insulin‐producing beta cells of the pancreas, yet the antigens that drive this response have been difficult to define. This review traces the id...
02
DOI 10.1111/imr.70182
Open DOI
Oxidative Stress, Post‐Translational Modifications, and Type 1 Diabetes Pathogenesis
Type 1 diabetes (T1D) is an organ‐specific autoimmune disease that destroys insulin‐producing beta‐cells. Insulin is a vital hormone for regulating glycemia that often undergoes post‐translational modifications during processing from pre...
03
DOI 10.1111/imr.70184
Open DOI
Entering the Age of Specificity in T Cell Immunity
The discovery of MHC restriction, the resolution of peptide–MHC structure, and the identification of the T cell receptor gene quickly centered epitope‐level specificity as a core concern of T cell biology. Epitope mapping culminated in f...
04
DOI 10.1111/imr.70183
Open DOI
Beta Cell Stress to Epitope Diversification: Post‐Translationally Modified Antigens in Type 1 Diabetes
Pancreatic beta cells are active participants in the loss of immune tolerance because their high secretory demand creates exceptional vulnerability to inflammatory exposure, endoplasmic reticulum (ER) stress, calcium dysregulation, and r...
05
DOI 10.1111/imr.70181
Open DOI
Human IgD + IgM − Memory B Cells and Their Possible Impact on Mucosal Homeostasis and Tolerance
This review discusses evidence indicating that the human nasopharyngeal mucosa includes a heterogeneous subset of memory B cells expressing IgD but not IgM. Such IgD + IgM − memory (IgD‐ME) B cells differentiate from germinal center B ce...
06
DOI 10.1111/imr.70180
Open DOI
Building an Interleukin‐10‐Producing Identity: How B Cells Acquire Interleukin‐10 Competence and How Plasma Cell Differentiation Consolidates This Program
Interleukin (IL)‐10‐producing B cells represent an archetype for the regulatory functions of B cells. Here, we review the cell‐surface receptors and transcriptional regulators that control IL‐10 expression in B cells and enable their reg...
07
DOI 10.1111/imr.70179
Open DOI
Discovery of Post‐Translationally Modified Epitopes in Autoimmunity: Someone Has to Fish so Everyone Can Eat
Post‐translational modifications (PTMs) chemically diversify the ~20,000 genomically encoded proteins into millions of functional variants, and this diversity has profound consequences for T cell‐mediated immune responses. PTM‐reactive T...
08
DOI 10.1111/imr.70132
Open DOI
Issue Information
09
DOI 10.1111/imr.70170
Open DOI
BAFF and APRIL Receptors in B Cell Immunity and Autoimmunity
BAFF and APRIL are TNF superfamily proteins that bind to BAFFR, TACI and BCMA, members of the TNF receptor superfamily. These proteins have both unique and overlapping roles in B cell development and survival and are major therapeutic ta...
10
DOI 10.1111/imr.70166
Open DOI
Complement Activation by Post‐Translationally Modified Proteins: Links to Chronic Inflammation and Autoimmunity
Post‐translational modifications (PTMs) of proteins are essential to maintain homeostasis as many cellular processes rely on reversible PTMs. However, several PTMs, particularly irreversible PTMs in the extracellular space, can contribut...
Bibliographical & Catalog Notes
Editor: 33- G. Möller.
Also issued online.
Vol. 54 (1981)19921022 Notice not published Printed on acid-free paper