Journal profile

Hämostaseologie

Publisher: Thieme
ISSN0720-9355 | 0720-9335 | 2567-5761 FormatPrint StatusCurrently indexed

Journal Citation Impact

Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.

Impact Factor
2.49
Citation rate 0.85 274 total citations
h5-index 23 h5 depth signal
Cited share 24.1% 78 of 324
Total citations 274 Citation footprint
Metric signal 2.25 External metric anchor
Self-citation 4.0% 11 self-citations

Citations Trend

Monthly Publications Cadence

Citing Geographic Demographics

Germany 576
Switzerland 111
Austria 96
United States 67
Italy 42

Citing Fields of Research

Hematology 177
Cardiology 32
Clinical Laboratory Science 22
Immunology 15
Vascular Disorders 8

Publishing Activity & Reach

Volume, recency, access pattern, and citing-community signals from the journal analytics record.

All-time article footprint 2,637 Articles tracked across the journal lifetime
Indexed publication set 324 78 cited publications
Last 12 months 249 24.9 publications/month
Open access share 5.6% 14 OA publications in the last year
Leading citing country Germany 576 citing publications
Leading citing field Hematology 177 citing publications
Latest tracked publication 7 October 2026 10 recent records available

Coverage & Indexing History

MEDLINE
v22n1, Feb. 2002-
PubMed
v22n1, Feb. 2002-
Index medicus
Active Coverage

Research Focus & Classification

Indexed Subjects

Hematology

Broad Categories

Hematology

MeSH Descriptors

Hemostasis

Latest Publications

Recent article records with DOI-first linking and condensed abstracts.

02
Rebekka Wiedenhoefer, Doris Boeckelmann, Hannah Glonnegger et al.
Platelet-type von Willebrand disease (PT-VWD) is a rare thrombocytopathy caused by gain-of-function variants in GP1BA, leading to increased binding of von Willebrand factor (VWF). Because its phenotype closely resembles VWD type 2B (VWD ...
03
Nina Cooper, Ulrich J. Sachs
Immune thrombocytopenia (ITP) is an acquired autoimmune disorder characterized by increased platelet destruction and impaired platelet production, resulting in isolated thrombocytopenia. Despite advances in understanding disease pathophy...
04
Jinxiong Jiang, Xiaoying Lü, Ziyan Wei et al.
Background Fibrinogen, encoded by FGA, FGB, and FGG, is essential for blood coagulation. Mutations in these genes cause congenital fibrinogen disorders, leading to bleeding and thrombotic complications. The Bβ chain is the rate-limiting ...

Bibliographical & Catalog Notes

Also issued online.

Text in German; table of contents in English and German.