Impact Factor
2.90
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
2.31
84 total citations
h5-index
13
h5 depth signal
Cited share
71.8%
28 of 39
Total citations
84
Citation footprint
Metric signal
3.14
External metric anchor
Self-citation
0.0%
0 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
China
147
United States
53
Japan
23
South Korea
14
Germany
12
Citing Fields of Research
Respiratory Support
104
Immunology
68
Lung Cancer
67
Allergies
59
Auto-Inflammatory Disorders
40
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
2,140
Articles tracked across the journal lifetime
Indexed publication set
39
28 cited publications
Last 12 months
19
1.7 publications/month
Open access share
100.0%
19 OA publications in the last year
Leading citing country
China
147 citing publications
Leading citing field
Respiratory Support
104 citing publications
Latest tracked publication
23 September 2026
10 recent records available
Coverage & Indexing History
v1n1,Mar. 1980-
v1n1,Mar. 1980-
Active Coverage
Research Focus & Classification
Indexed Subjects
Respiratory Support
Immunology
Noncoding RNA
Physiology
Auto-Inflammatory Disorders
Allergies
Broad Categories
Pulmonary Medicine
Respiratory Support
Inflammatory Bowel Diseases & Infections
Noncoding RNA
Allergies
Cystic Fibrosis
Transplantation
MeSH Descriptors
Lung Diseases
Lung
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
DOI 10.1080/01902148.2026.2738090
Open DOI
Statement of Retraction
02
DOI 10.1080/01902148.2026.2704400
Open DOI
Mechanism of IH promoting pulmonary arterial hypertension through ROS-NLRP3 inflammasome mediated endothelial cell pyroptosis
Pulmonary arterial hypertension (PAH) refers to a common cardiovascular disease in which the mean pulmonary artery pressure (mPAP) is greater than 20 mmHg, pulmonary vascular resistance (PVR) is ≥3...
03
DOI 10.1080/01902148.2026.2721778
Open DOI
Correlation between ACE2 and monocrotaline-induced pulmonary arterial hypertension in rats
Pulmonary arterial hypertension (PAH) is a disease that is primarily characterized by pulmonary vasoconstriction and vascular remodeling.1 PAH ultimately progresses to right heart failure and death...
04
05
DOI 10.1080/01902148.2026.2707582
Open DOI
Correction
Article title: The carotid body role in oxygen sensing: Anatomy, neurovascular organization, and structural bases of chemotransductionAuthors: Ahmaed Baashar and Zaki AlsahafiJournal:Experimental L...
06
DOI 10.1080/01902148.2026.2694801
Open DOI
The carotid body role in oxygen sensing: Anatomy, neurovascular organization, and structural bases of chemotransduction
The carotid body is a small, highly vascularized organ located at the bifurcation of the common carotid artery and serves as the primary peripheral detector of arterial oxygen tension in mammals. Although its physiological role in ventil...
07
DOI 10.1080/01902148.2026.2694260
Open DOI
The role of ABHD2 in cigarette smoke-induced skeletal muscle atrophy in chronic obstructive pulmonary disease mice: The mechanism of ABHD2 in skeletal muscle atrophy.
Background: Chronic obstructive pulmonary disease (COPD) is a major and increasing global health problem that results in progressive airway obstruction. Cigarette smoke (CS) exposure, a major cause for COPD, induces mitochondrial damage,...
08
DOI 10.1080/01902148.2026.2664336
Open DOI
Mechanistic study on the role of NCSTN in regulating the differentiation of airway epithelial basal cells into ciliated cells following CSE exposure
Chronic obstructive pulmonary disease (COPD) is a respiratory condition characterized by chronic and irreversible airflow limitation. It is the third leading cause of death worldwide, with long-ter...
09
DOI 10.1080/01902148.2026.2645470
Open DOI
The downregulation of RAB10 by miR-574-5p alleviates the inflammatory response in neonatal respiratory distress syndrome
The diagnosis of neonatal respiratory distress syndrome NRDS relies on progressive breathing difficulties after birth, abnormal blood oxygen levels, and typical chest X-ray images. However, this diagnostic method has insufficient specifi...
10
DOI 10.1080/01902148.2026.2649073
Open DOI
Copper homeostasis dysregulation and cuproptosis in respiratory diseases
Cuproptosis, a recently identified form of regulated cell death, relies on accumulated copper (Cu) ions and a disrupted mitochondrial respiratory chain. The role of cuproptosis in various diseases, especially respiratory diseases of pulm...
Bibliographical & Catalog Notes
Imprint varies.
Also issued online.
Vol. 18, no. 1 (JAN-MAR 1992)19931110 Notice published Printed on acid-free paper that meets the full ANSI standard for Permanent Paper