Journal profile

Epigenetics & chromatin

Publisher: BioMed Central
ISSN1756-8935 FormatOnline StatusCurrently indexed

Journal Citation Impact

Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.

Impact Factor
3.98
Citation rate 5.99 491 total citations
h5-index 36 h5 depth signal
Cited share 86.4% 76 of 88
Total citations 491 Citation footprint
Metric signal 8.37 External metric anchor
Self-citation 1.6% 8 self-citations

Citations Trend

Monthly Publications Cadence

Citing Geographic Demographics

United States 257
China 96
Germany 63
United Kingdom 39
Japan 39

Citing Fields of Research

Genetics & Genomics 531
Cytogenetics 166
Cell Biology 79
Noncoding RNA 60
DNA Damage 58

Publishing Activity & Reach

Volume, recency, access pattern, and citing-community signals from the journal analytics record.

All-time article footprint 1,057 Articles tracked across the journal lifetime
Indexed publication set 88 76 cited publications
Last 12 months 72 6.0 publications/month
Open access share 100.0% 72 OA publications in the last year
Leading citing country United States 257 citing publications
Leading citing field Genetics & Genomics 531 citing publications
Latest tracked publication 9 October 2026 10 recent records available

Coverage & Indexing History

MEDLINE
v9, 2016-
PubMed
v1, 2008-
PMC
Active Coverage

Research Focus & Classification

Indexed Subjects

Genetics & Genomics

Broad Categories

Biochemistry Genetics Genetics & Genomics

MeSH Descriptors

Chromatin Epigenesis, Genetic

Latest Publications

Recent article records with DOI-first linking and condensed abstracts.

01
Yong Hwee Eddie Loh, Robert Morgan, Devika Singh et al.
Background: DNA methylation is a key epigenetic mechanism of sex chromosome regulation. However, DNA methylation studies often exclude X chromosomes. Moreover, widely used DNA methylation assays interrogate biased subsets of CpGs, rather...
07
Steven Henikoff, Robert N. Eisenman, Kami Ahmad
Global upregulation of transcription, or hypertranscription, is observed in most human cancers, driven by the action of oncogene-encoded transcription factors (TFs). TF interactions with RNA Polymerase II (Pol II) are mediated by kinases...
10
Vilma Canfjorden, Malin Lindén, Tobias Österlund et al.
Background: Approximately 20% of all cancers contain mutations in genes coding for components of the SWI/SNF chromatin remodeling complex, which exists in three main subtypes: cBAF, PBAF, and GBAF. Mutations in SWI/SNF genes may disrupt ...

Bibliographical & Catalog Notes

Description based on: 1:1 (27 October 2008); title from journal home page (publisher's Web site, viewed Oct. 27, 2008).