Impact Factor
3.28
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
2.75
362 total citations
h5-index
24
h5 depth signal
Cited share
74.1%
100 of 135
Total citations
362
Citation footprint
Metric signal
2.53
External metric anchor
Self-citation
5.0%
18 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
China
286
United States
201
Italy
92
India
79
Spain
67
Citing Fields of Research
Neurodegenerative Disorders
1,041
Neurosciences
151
Aging & Longevity
36
Genetics & Genomics
34
Noncoding RNA
25
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
2,319
Articles tracked across the journal lifetime
Indexed publication set
135
100 cited publications
Last 12 months
106
8.8 publications/month
Open access share
1.9%
2 OA publications in the last year
Leading citing country
China
286 citing publications
Leading citing field
Neurodegenerative Disorders
1,041 citing publications
Latest tracked publication
12 October 2026
10 recent records available
Coverage & Indexing History
v1n1, Feb. 2004-
v1n1, Feb. 2004-
Active Coverage
Research Focus & Classification
Indexed Subjects
Neurodegenerative Disorders
Broad Categories
Neurology
Neurodegenerative Disorders
MeSH Descriptors
Alzheimer Disease
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
DOI 10.2174/0115672050525938260923072703
Open DOI
Transitioning from Clinical Trial to Clinical Practice for Long-Term Lecanemab Treatment in Early Alzheimer’s Disease: Perspectives from a Cohort Study from the Alzheimer’s Research and Treatment Center
Introduction/Background: Lecanemab, an anti-amyloid monoclonal antibody, significantly reduces amyloid markers and slows clinical decline in early Alzheimer’s disease (AD) clinical trials. We assessed our single-center experience with le...
02
DOI 10.2174/0115672050512147260928102334
Open DOI
Global Dynamics and Emerging Trends in Alzheimer's Disease and Apoptosis from 2007 to 2025: A Bibliometric Analysis of Scholarly Evolution
Introduction: To conduct the first systematic bibliometric analysis of global research on the relationship between Alzheimer's disease (AD) and apoptosis from 2007 to 2025. Methods: This study utilized the Web of Science Core Collection ...
03
DOI 10.2174/0115672050502048260925094031
Open DOI
Tetrachloro-1,4-benzoquinone Exerts Neurotoxicity through Neuroinflammation and the TLR4 Signaling Pathway, which is a Risk Factor for Alzheimer's Disease: An Integrated Network and Mendelian Randomization Analysis
Introduction: Research has found that the disinfection byproduct Tetrachloro-1,4- Benzoquinone (TCBQ) in drinking water increases the risk of Alzheimer's Disease (AD). However, the mechanism by which TCBQ increases the risk of AD remains...
04
DOI 10.2174/0115672050504771260920194040
Open DOI
Genetic Evidence of Association Between Plasma Cathepsin L2 and Alzheimer’s Disease: A Mendelian Randomization Study
Background: Recent studies have suggested cathepsins, lysosomal cysteine proteases, are associated with Alzheimer’s DISEASE (AD). This study conducted Mendelian Randomization (MR) analysis to systematically assess the causal effect of ge...
05
DOI 10.2174/0115672050513704260903053055
Open DOI
Vitamin C versus Alzheimer’s Disease. Commentary on the Effect of Ascorbic Acid on the Transgenic Drosophila Expressing Human Aβ-42 in the Neurons
A recent article by Siddique et al. published in Current Alzheimer's Research suggests that vitamin C may have a neuroprotective effect in Alzheimer's disease, which inspired us to write this commentary. In this commentary, we focus on t...
06
DOI 10.2174/0115672050493089260903062914
Open DOI
Immunity and Inflammation in Alzheimer's Disease: A Narrative Review on Mechanisms and Therapeutic Prospects
Alzheimer’s Disease (AD) is an age-related neurodegenerative disorder of the Central Nervous System (CNS), characterized by amyloid-β (Aβ) deposition and Neurofibrillary Tangles (NFTs) caused by the hyperphosphorylation of tau protein. N...
07
DOI 10.2174/0115672050503391260907045733
Open DOI
Basic Research on the Lymphatic System and Exploratory Research in LVA Surgery for Alzheimer's Disease
Introduction: Alzheimer's Disease (AD) is a common neurodegenerative disorder characterized by progressive cognitive decline. Its characteristic pathological features include Aβ plaque deposition and abnormal tau protein aggregation. Bio...
08
DOI 10.2174/0115672050489243260827065622
Open DOI
Novel Amyloid-β Aggregation Inhibitors Based on Phenylalanine Structure
Introduction/Objective: Alzheimer’s disease (AD) is characterized by amyloid-β (Aβ) aggregation, making its inhibition a promising therapeutic strategy. This study evaluated the effects of two phenylalanine derivatives on Aβ aggregation ...
09
DOI 10.2174/0115672050468773260821110356
Open DOI
Altered Epoxyeicosatrienoic Acid Signaling in Advanced-Stage Alzheimer’s Disease (Investigation of Epoxyeicosatrienoic Acid Levels in Patients with Advanced Alzheimer’s Disease)
Introduction: The study aimed to compare plasma Epoxyeicosatrienoic Acid (EET) isomers (5,6-EET, 8,9-EET, 11,12-EET, and 14,15-EET) in patients with advanced-stage late-onset Alzheimer’s Disease (AS-LOAD) versus healthy controls matched ...
10
DOI 10.2174/0115672050478591260821105132
Open DOI
Decoding Frontotemporal Dementia: Genes, Proteins and Prospects for Precision Medicine
Frontotemporal Dementia is an umbrella term for several neurodegenerative disorders that have a similar presentation pattern, which is accompanied by neural damage and brain tissue loss in the frontal and temporal regions. Recent studies...
Bibliographical & Catalog Notes
Latest issue consulted: Volume 15, issue 7 (2018).
Also issued online.