Impact Factor
2.78
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
2.03
142 total citations
h5-index
13
h5 depth signal
Cited share
63.4%
45 of 71
Total citations
142
Citation footprint
Metric signal
1.59
External metric anchor
Self-citation
3.5%
5 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
United States
150
China
92
India
30
Brazil
17
Japan
11
Citing Fields of Research
Immunology
234
Noncoding RNA
21
Immunotherapy
20
Musculoskeletal Disorders
18
Allergies
17
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
906
Articles tracked across the journal lifetime
Indexed publication set
71
45 cited publications
Last 12 months
26
26.0 publications/month
Open access share
23.1%
6 OA publications in the last year
Leading citing country
United States
150 citing publications
Leading citing field
Immunology
234 citing publications
Latest tracked publication
1 January 2026
10 recent records available
Coverage & Indexing History
v1issue2,May 1980-
v1issue2,May 1980-
Active Coverage
Research Focus & Classification
Indexed Subjects
Immunology
Broad Categories
Allergy and Immunology
Noncoding RNA
Immunology
Cancer, Autophagy & Apoptosis
Musculoskeletal Disorders
Osteopathies
Respiratory Support
MeSH Descriptors
Immunity
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
DOI 10.1615/critrevimmunol.2026065461
Open DOI
Tumor-Infiltrating B Cells: Implications for Immunotherapy Response and Prognosis in Non-Small Cell Lung Cancer
02
DOI 10.1615/critrevimmunol.2026065812
Open DOI
Inhibition of METTL3-mediated STAT3 m6A modification enhances the sensitivity of head and neck squamous cell carcinoma to FGFR inhibitor AZD4547
03
DOI 10.1615/critrevimmunol.2026065430
Open DOI
Reprogramming Macrophages for Breast Cancer Therapy
04
DOI 10.1615/critrevimmunol.2026065366
Open DOI
Circular RNA circ_0000064 Promotes Epithelial-Mesenchymal Transition of Lung Adenocarcinoma by Sponging miR-532-3p to Upregulate FOXM1 Expression
05
06
DOI 10.1615/critrevimmunol.2026063398
Open DOI
Targeting Substance P–NK1R Signaling: Emerging Immunological Mechanisms and Therapeutic Promise of Substance P Inhibitors
Substance P (SP), a highly conserved neuropeptide of the tachykinin family, serves as a critical mediator of neuroimmune communication and host defense. Although traditionally associated with pain transmission, vasodilation, and neurogen...
07
DOI 10.1615/critrevimmunol.2026065692
Open DOI
Ciprofol combined with CA1 overexpression suppresses colorectal cancer tumorigenesis via p53 signaling pathway
08
DOI 10.1615/critrevimmunol.2026065687
Open DOI
Oxymatrine Attenuates Lung Adenocarcinoma Tumorigenesis via METTL3-Mediated the m6A Modification of KAT2A
Background: Oxymatrine executes a therapeutic effect on malignant phenotype tumor cells; nevertheless, the mechanisms of oxymatrine in lung adenocarcinoma (LUAD) require further exploration. Here, the aims of our study are to unveil the ...
09
DOI 10.1615/critrevimmunol.2026062734
Open DOI
NK cells in endocrine autoimmune disorders, current knowledge and implications for therapeutic strategies
Natural killer (NK) cells play critical roles in innate immunity and in shaping adaptive immune responses. Recent studies reveal their dual role in autoimmunity, either with a protective role by eliminating autoreactive cells or as contr...
10
DOI 10.1615/critrevimmunol.2026064384
Open DOI
Towards a Unified Multidimensional HIV Cure: Integrating Virologic, Clinical, and Immunologic Perspectives
Despite major advances in antiretroviral therapy (ART), a definitive cure for human immunodeficiency virus (HIV) remains elusive. This review aims to propose a unified, multidimensional framework for HIV cure that integrates virologic, i...
Bibliographical & Catalog Notes
Also issued online.
Vol. 1, no. 2 (MAY 1980)19911012 Notice not published Printed on acid-free paper