Impact Factor
4.33
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
7.91
1,594 total citations
h5-index
55
h5 depth signal
Cited share
85.8%
182 of 212
Total citations
1,594
Citation footprint
Metric signal
7.03
External metric anchor
Self-citation
0.9%
15 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
United States
472
China
448
United Kingdom
211
Australia
172
Brazil
92
Citing Fields of Research
Immunology
276
Cardiology
266
Kidney Diseases
229
Diabetes
228
Physiology
152
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
19,157
Articles tracked across the journal lifetime
Indexed publication set
212
182 cited publications
Last 12 months
167
13.9 publications/month
Open access share
88.0%
147 OA publications in the last year
Leading citing country
United States
472 citing publications
Leading citing field
Immunology
276 citing publications
Latest tracked publication
16 October 2026
10 recent records available
Coverage & Indexing History
v57n1, July 1979-
v57n1, July 1979-
Active Coverage
Research Focus & Classification
Indexed Subjects
Immunology
Diabetes
Kidney Diseases
Cardiology
Noncoding RNA
Mitochondria
Issuing & Sponsoring Bodies
Contributor
Biochemical Society (Great Britain)
Contributor
Medical Research Society (Great Britain)
Contributor
International Society of Hypertension
Broad Categories
Medicine
Science
Urology
Molecular Pharmacology
Diabetes
Cancer, Autophagy & Apoptosis
Noncoding RNA
Physiology
Cardiology
MeSH Descriptors
Biochemistry
Medicine
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
DOI 10.1042/cs20260162
Open DOI
Sex-Specific Adaptations in Placental Mitochondrial Biogenesis and Function in babies Born with Critical Congenital Heart Disease
Congenital heart disease (CHD) is the leading cause of birth defects; most critical (c)CHD newborns will develop neurodevelopmental disorders. Abnormal placental development may be involved, as genetics explains only 30% of sporadic CHD ...
02
DOI 10.1042/cs20250834
Open DOI
Metabolic gatekeepers shape immune cell function in rheumatoid arthritis
Immune cell function is tightly coupled to cellular metabolism, with glycolysis, oxidative phosphorylation, and amino acid and lipid metabolism governing activation, differentiation, survival, and effector responses. In rheumatoid arthri...
03
DOI 10.1042/cs20260881
Open DOI
Ten–eleven translocation 3 activates the TGF-β-like ligand Scube3 to promote renal fibrosis: mechanism and translational potential
Resident renal fibroblasts are a predominant source for myofibroblasts that produce ECM to contribute to renal fibrosis in the course of chronic kidney disease (CKD). The mechanism whereby fibroblast–myofibroblast transition (FMyT) is dr...
04
DOI 10.1042/cs20261535
Open DOI
E2F1 down-regulation promotes lipid droplet accumulation and endometriosis development through impaired MGLL-mediated lipid mobilization
Endometriosis is a common gynecological disease in which lipid metabolic dysregulation has been increasingly implicated. However, the specific metabolic changes in ectopic lesions and stromal cells remain poorly understood. It is also un...
05
DOI 10.1042/cs20261713
Open DOI
Type 2 angiotensin II receptor (AT2R) deficiency exacerbates cardiac senescence and fibrosis in aging mice
Cardiac aging increases susceptibility to cardiovascular diseases. Cellular senescence may be involved in the development of age-related cardiac diseases. Extensive evidence derived from both clinical and experimental studies suggests th...
06
DOI 10.1042/cs20261842
Open DOI
Post-ischemic inhibition of the mitochondrial Ca2+ uniporter with Ru265 protects in ex vivo and in vivo models of cerebral ischemia
Brain ischemia is a leading cause of morbidity and mortality in the aging population, yet no effective neuroprotective interventions currently exist. This therapeutic gap stems from two key challenges: the inability to deliver neuroprote...
07
DOI 10.1042/cs20262448
Open DOI
Deletion of Nephrocystin-4 Exacerbates Kidney and Cardiac Function in Mice Cardiorenal Syndrome Model via the Interaction with Itch E3 ligase
Cardiorenal syndrome (CRS), characterized by cardiac and renal dysfunction, is an increasingly common health issue with a high mortality rate. Nephrocystin 4 (NPHP4) is the major reason for end-stage renal disease in children. We have re...
08
DOI 10.1042/cs20261278
Open DOI
Aberrant absorptive enterocyte differentiation is associated with altered ion transport and chronic inflammatory diahrea in IBD patients
Diarrhea is a hallmark of Crohn’s disease (CD) and ulcerative colitis (UC), often linked to a leaky epithelial barrier and an impaired fluid and electrolyte absorption. This study investigates whether a defect in enterocyte differentiati...
09
10
Bibliographical & Catalog Notes
Print issues ceased with v. 132 (2018).
Sequentially numbered supplements accompany some vols.
Supplements for 1979-1982 consist of the proceedings of the 6th-9th meeting of the International Society of Hypertension.
Also issued online.
Vol. 73, no. 1 (1987)19920824 Notice not published Printed on acid-free paper