Impact Factor
3.83
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
4.13
1,007 total citations
h5-index
44
h5 depth signal
Cited share
82.2%
212 of 258
Total citations
1,007
Citation footprint
Metric signal
4.13
External metric anchor
Self-citation
1.5%
15 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
United Kingdom
273
China
257
United States
231
Italy
96
Germany
93
Citing Fields of Research
Immunology
972
Musculoskeletal Disorders
226
Auto-Inflammatory Disorders
163
Hematology
147
Allergies
81
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
9,297
Articles tracked across the journal lifetime
Indexed publication set
258
212 cited publications
Last 12 months
72
12.0 publications/month
Open access share
43.1%
31 OA publications in the last year
Leading citing country
United Kingdom
273 citing publications
Leading citing field
Immunology
972 citing publications
Latest tracked publication
16 October 2026
10 recent records available
Coverage & Indexing History
v1n1, Jan. 1966-
v1n1, Jan. 1966-
Active Coverage
Active Coverage
Research Focus & Classification
Indexed Subjects
Immunology
Issuing & Sponsoring Bodies
Contributor
British Society for Immunology
Broad Categories
Allergy and Immunology
Immunology
Hematology
Musculoskeletal Disorders
Auto-Inflammatory Disorders
Allergies
Digestive Disorders
MeSH Descriptors
Allergy and Immunology
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
DOI 10.1093/cei/uxag064
Open DOI
Dysregulated complement activation in COVID-19: The Role of Soluble Complement Inhibitors
In viral infections, complement activation can contribute to viral clearance, but excessive activation, as observed in severe COVID-19, may drive immunopathology. While much is known about complement-activating factors in COVID-19, insig...
02
DOI 10.1093/cei/uxag062
Open DOI
Diagnosing primary immunodeficiencies using protein-based screening assays
Primary immunodeficiencies (PIDs), now more commonly referred to as inborn errors of immunity (IEIs), are a diverse group of inherited conditions characterized by immune system defects, leading to recurrent infections, immune dysregulati...
03
DOI 10.1093/cei/uxag060
Open DOI
Precision targeting and alternative effector cells: Revolutionizing CAR-based therapy for T-cell malignancies
T-cell malignancies are highly heterogeneous and associated with high relapse rate and poor prognosis. While chimeric antigen receptor-T (CAR-T) cell therapy has achieved remarkable success in relapsed or refractory B-cell malignancies, ...
04
DOI 10.1093/cei/uxag054
Open DOI
Epitope-Specific Thyroid Peroxidase Antibodies Reveal Functional Heterogeneity Within a Patient-Derived Monoclonal Repertoire
Introduction: Thyroid peroxidase (TPO) autoantibodies are defining features of autoimmune thyroid disease (AITD), yet the relationship between epitope specificity and antibody function remains incompletely understood. Methods: In this st...
05
06
DOI 10.1093/cei/uxag040
Open DOI
The silent regulators: Non-Coding RNAs and EBV at the crossroads of Lupus pathogenesis.
Systemic lupus erythematosus (SLE) is a heterogeneous autoimmune disease driven by immune dysregulation, chronic inflammation, and loss of immune tolerance. Increasing evidence suggests that non-coding RNAs (ncRNAs), including microRNAs,...
07
DOI 10.1093/cei/uxag041
Open DOI
Helios expression in naïve CD4+ T cells decreases from neonates to older adults
Introduction: Fetuses, neonates, and infants are vulnerable to infectious diseases. At the same time, they are exposed to many harmless antigens; therefore, suppressing unnecessary immune responses to maintain immune tolerance is a ratio...
08
DOI 10.1093/cei/uxag038
Open DOI
Could we discuss the molecular signature of immune dysregulation?
Introduction: Immune dysregulation (ID), defined as aberrant immune activation or suppression, may result in autoimmune or inflammatory disorders, lymphoproliferative malignancies, and allergic diseases. Apoptosis, nuclear factor-κB (NFκ...
09
DOI 10.1093/cei/uxag039
Open DOI
The many faces of cytokine storm syndrome: immunopathogenic mechanisms and clinical implications for a better patient management
Cytokine storm syndrome is a life-threatening hyperinflammatory condition characterized by excessive and dysregulated immune activation, leading to multiorgan dysfunction and poor clinical outcomes if not promptly recognized and adequate...
10
DOI 10.1093/cei/uxag037
Open DOI
PD-1 Engagement and Circulating Th17 Effector Activity During Secukinumab Treatment in Plaque Psoriasis
Introduction: Plaque psoriasis (PP) is a chronic immune-mediated inflammatory disease driven by dysregulation of the IL-23/Th17/IL-17 axis. Although IL-17A targeted biologics achieve robust clinical efficacy, how cytokine blockade is acc...
Bibliographical & Catalog Notes
Also issued online.
Official journal of the British Society for Immunology.
Will not microfilm 2005225
Vol. 39, no. 1 (1980)19920818 Notice not published Printed on acid-free paper