Impact Factor
4.83
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
14.72
4,553 total citations
h5-index
95
h5 depth signal
Cited share
95.4%
312 of 327
Total citations
4,553
Citation footprint
Metric signal
14.55
External metric anchor
Self-citation
0.7%
30 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
China
951
United States
584
Germany
238
Italy
194
United Kingdom
172
Citing Fields of Research
Cancer, Autophagy & Apoptosis
1,030
Immunology
449
Cell Biology
397
Cancer & Development
350
Noncoding RNA
194
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
5,892
Articles tracked across the journal lifetime
Indexed publication set
327
312 cited publications
Last 12 months
285
23.8 publications/month
Open access share
53.3%
152 OA publications in the last year
Leading citing country
China
951 citing publications
Leading citing field
Cancer, Autophagy & Apoptosis
1,030 citing publications
Latest tracked publication
19 October 2026
10 recent records available
Coverage & Indexing History
v5n1, Jan. 1998-
v5n1, Jan. 1998-
Active Coverage
Active Coverage
Research Focus & Classification
Indexed Subjects
Cancer, Autophagy & Apoptosis
Immunology
Cancer & Development
Cell Biology
DNA Damage
Broad Categories
Cell Biology
Cancer, Autophagy & Apoptosis
MeSH Descriptors
Cell Death
Cell Differentiation
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
02
DOI 10.1038/s41418-026-01870-y
Open DOI
PABPC1 dictates castration sensitivity in prostate cancer via orchestrating ferroptosis resistance and CD8(+) T-cell dysfunction
Resistance to next-generation androgen receptor antagonists represents a major clinical challenge in prostate cancer (PCa), underscoring the need to define its underlying mechanisms. Here, through an integrated analysis of multiple cohor...
03
DOI 10.1038/s41418-026-01891-7
Open DOI
A chemical-genetic system for calreticulin exposure on cancer cells reveals immune-dependent antitumor effects
Premortem exposure of calreticulin (CALR) on stressed cancer cells is a cardinal damage-associated molecular pattern that licenses anticancer immunity, yet its immunological sufficiency has remained difficult to define because it normall...
04
DOI 10.1038/s41418-026-01885-5
Open DOI
USP25 augments XIAP-mediated signaling to safeguard intestinal homeostasis
05
DOI 10.1038/s41418-026-01889-1
Open DOI
Death domain-mediated higher-order oligomerization of RIPK1 drives TNF-induced signaling
RIPK1 paradoxically orchestrates pro-survival and pro-death pathways through its scaffolding and kinase activities. Downstream of TNFR1, RIPK1 initiates the formation of two distinct multiprotein complexes: complex I for cell survival an...
06
DOI 10.1038/s41418-026-01886-4
Open DOI
Ubiquitination of LMP-1 by UFD-2 regulates apoptotic cell clearance
Apoptotic cell clearance, or efferocytosis, is essential for tissue homeostasis and preventing inflammation in multicellular organisms. Through a genome-wide RNAi screen targeting 169 E3 ubiquitin ligases in Caenorhabditis elegans, we id...
07
DOI 10.1038/s41418-026-01880-w
Open DOI
nHOTAIRM1 scaffolds ANXA11-dependent assemblies to drive axonal mRNA localization in human motor neurons
Long noncoding RNAs (lncRNAs) are well recognized as regulators of neuronal development and function, yet their integration into canonical axonal transport pathways, an essential component of spatial gene regulation in highly polarized c...
08
DOI 10.1038/s41418-026-01881-9
Open DOI
Synergistic role of TFEB/TFE3 and mTORC1 in kidney cystogenesis and tumorigenesis
Birt-Hogg-Dubé (BHD) and Tuberous Sclerosis (TSC) are inherited cancer syndromes associated with kidney cystogenesis and tumorigenesis and caused by mutations of the folliculin (FLCN) and TSC1/2 genes, respectively. We and others previou...
09
DOI 10.1038/s41418-026-01856-w
Open DOI
FDX1-short proteoform induces caspase-independent cell death in tumor cells
Cell death is a crucial process for the maintenance of physiological functions in organisms. In recent years, cuproptosis, a novel form of cell death induced by copper overload in mitochondria, has emerged as a critical regulatory mechan...
10
DOI 10.1038/s41418-026-01882-8
Open DOI
Efferocytosis triggered proteomic reprogramming of macrophages reveals CD97 as a novel apoptotic receptor
During the daily process of healthy cellular turnover in the human body, billions of cells undergo apoptosis. These cells are removed by phagocytic cells, namely macrophages through a process known as efferocytosis, which triggers a casc...
Bibliographical & Catalog Notes
Issues for Jan. 2005- have title: Cell death & differentiation.
Title from cover.
Vol. 1 complete in 2 issues.
Also issued online.
Affiliated with: Cell death discovery 2058-7716 (DLC) 2015243428 (OCoLC)926100762.
Vol. 4, no. 1 (JAN 1997)19970722 Notice published Printed on acid-free paper