Impact Factor
4.70
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
22.06
2,861 total citations
h5-index
73
h5 depth signal
Cited share
97.1%
132 of 136
Total citations
2,861
Citation footprint
Metric signal
26.77
External metric anchor
Self-citation
2.5%
71 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
China
378
United States
251
Germany
31
Australia
30
United Kingdom
27
Citing Fields of Research
Osteopathies
250
Musculoskeletal Disorders
236
Cell Differentiation & Development
183
Stem Cell Biology
95
Cell Biology
41
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
712
Articles tracked across the journal lifetime
Indexed publication set
136
132 cited publications
Last 12 months
126
10.5 publications/month
Open access share
100.0%
126 OA publications in the last year
Leading citing country
China
378 citing publications
Leading citing field
Osteopathies
250 citing publications
Latest tracked publication
16 October 2026
10 recent records available
Coverage & Indexing History
v11, 2023-
v1n1, Mar. 2013-
Active Coverage
Research Focus & Classification
Indexed Subjects
Musculoskeletal Disorders
Osteopathies
Cell Differentiation & Development
Stem Cell Biology
Issuing & Sponsoring Bodies
sponsoring body
Sichuan da xue
Contributor
International Chinese Hard Tissue Society
Broad Categories
Metabolism
Orthopedics
Musculoskeletal Disorders
Osteopathies
Cancer & Development
Cell Differentiation & Development
Cancer, Autophagy & Apoptosis
MeSH Descriptors
Bone Diseases
Bone and Bones
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
DOI 10.1038/s41413-026-00587-5
Open DOI
Integrated stress signaling pathways promote adaptive reprogramming for chondrocyte survival in Metaphyseal Chondrodysplasia Type Schmid
Collagen X, encoded by the Col10a1 gene, is expressed specifically by hypertrophic chondrocytes (HCs) in the cartilage. Mutations of the Col10a1 gene in humans, which increase the endoplasmic reticulum (ER) stress in HCs, lead to metaphy...
02
DOI 10.1038/s41413-026-00589-3
Open DOI
15-PGDH inhibition in osteoarthritis: PGE2 skeletal interoception, cartilage regeneration and compartment-managed disease modification
Osteoarthritis is a whole-joint disease in which cartilage loss, synovial inflammation, subchondral bone remodelling and pain-related neural changes co-evolve, yet structural improvement does not reliably translate into pain relief. This...
03
DOI 10.1038/s41413-026-00565-x
Open DOI
Bi-allelic EPHA2 mutations cause dysosteosclerosis by inhibiting osteoclastogenesis and enhancing osteoblastogenesis
Erythropoietin-producing hepatocellular (EPH) receptors, the largest receptor tyrosine kinase family, have been shown to play a crucial role in normal physiological processes and pathologies. EPHA2, encoded by EPHA2 gene, a member of the...
04
DOI 10.1038/s41413-026-00567-9
Open DOI
C-type lectin receptors: bone remodeling and therapeutic potential
Osteoimmunology explores the interplay between the skeletal system and the immune system. It focuses on the identification of shared signaling pathways and the mechanisms of mutual regulation. Bone homeostasis depends on the balance betw...
05
DOI 10.1038/s41413-026-00559-9
Open DOI
Biomaterial-mediated blood clot formation: a key to unlocking osteogenic synergy for bone regeneration
Bone hemorrhage presents serious clinical challenges due to its high morbidity and mortality. Autologous coagulation forms blood clots that both halt bleeding and provide a natural scaffold for bone repair. The dynamic formation and diss...
06
DOI 10.1038/s41413-026-00585-7
Open DOI
Multi-omic analysis of jaw-bone periosteum identifies the role of macrophage-derived SPP1 in Ctsk+Fmod+ periosteal cells
Periosteal cells (PCs) play a crucial role in bone regeneration. Currently, the spatial landscapes of jaw-bone periosteum during bone grafting remains unclear. In this study, we propose a modified guided bone regeneration (GBR) model and...
07
DOI 10.1038/s41413-026-00575-9
Open DOI
Spleen-derived circulating intercellular adhesion molecule 2 promotes osteosclerosis in chronic myelofibrosis
Mutations in the gene coding G6b-B, Mpig6b in mice, and MPIG6B in humans lead to myelofibrosis, progressive splenomegaly and osteosclerosis. Aberrant megakaryocyte function in bone marrow and spleen may affect bone cells through cell-cel...
08
DOI 10.1038/s41413-026-00582-w
Open DOI
Bone aging: a paradigm of multiscale degeneration and targeted rejuvenation
Bone aging is not merely a process of bone mass loss but rather a multi-level, networked degenerative phenomenon spanning molecular, cellular, tissue, and system levels. With the accelerating pace of global population aging, bone degener...
09
DOI 10.1038/s41413-026-00560-2
Open DOI
WDR23 prevents bone loss by promoting autophagic degradation of TRAF6 in osteoclastogenesis
Tumor necrosis factor receptor-associated factor 6 (TRAF6) is a pivotal adaptor molecule in the receptor activator of nuclear factor-κB (RANK) and its ligand (RANKL) signaling pathways, which are essential for osteoclastogenesis. In this...
10
DOI 10.1038/s41413-026-00581-x
Open DOI
Single-cell multi-omics defines H3K27me3 remodelling in intervertebral disc degeneration with implications for regenerative intervention
Utilizing key developmental cues and refining their orchestrating role in degeneration represents a promising strategy for understanding and treating intervertebral disc (IVD) degeneration, a major cause of chronic lower back pain. Here,...
Bibliographical & Catalog Notes
Online issue, viewed Aug. 15, 2013 for May 2013 identified as June 2013.
Also issued online.