Impact Factor
3.72
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
3.85
1,140 total citations
h5-index
40
h5 depth signal
Cited share
69.9%
248 of 355
Total citations
1,140
Citation footprint
Metric signal
4.36
External metric anchor
Self-citation
0.5%
6 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
United States
587
United Kingdom
173
Germany
156
France
110
Canada
105
Citing Fields of Research
Genetics & Genomics
442
Cell Biology
404
Evolutionary Genomics
149
Cell Differentiation & Development
134
DNA Damage
123
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
7,804
Articles tracked across the journal lifetime
Indexed publication set
355
248 cited publications
Last 12 months
114
9.5 publications/month
Open access share
64.0%
73 OA publications in the last year
Leading citing country
United States
587 citing publications
Leading citing field
Genetics & Genomics
442 citing publications
Latest tracked publication
9 October 2026
10 recent records available
Coverage & Indexing History
v3n1, 1985-
v3n1, 1985-
Active Coverage
Research Focus & Classification
Indexed Subjects
Cell Biology
Genetics & Genomics
Immunology
DNA Damage
Evolutionary Genomics
Cancer & Development
Issuing & Sponsoring Bodies
Contributor
International Council of Scientific Unions
Contributor
Company of Biologists
Broad Categories
Biology
Cell Biology
Molecular Biology
Genetics & Genomics
Stem Cell Biology
Cancer, Autophagy & Apoptosis
Cancer & Development
DNA Damage
Nucleic Acids Chemistry
MeSH Descriptors
Cell Biology
Growth
Molecular Biology
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
DOI 10.1002/bies.70188
Open DOI
Crosstalk in Silence: PIWIL/piRNA in ncRNA Hubs for Immune Gene Regulation and Epigenetic Antiviral Defense
Recent studies indicate that PIWI (P‐element‐induced WImpy testis) like proteins and their cognate PIWI‐interacting RNAs (piRNAs), initially identified in germline cells for protecting genomic stability by transposon silencing, also exer...
02
DOI 10.1002/bies.70194
Open DOI
The Partition Code: How RNA Modifications Set the Address of Transcripts Among Membraneless Compartments
Where a transcript goes among a cell's membraneless compartments—stress granules, P‐bodies, paraspeckles, nuclear speckles—is an addressable, rewritable property that this article proposes chemical modifications of RNA help write. The co...
03
DOI 10.1002/bies.70196
Open DOI
Biological Systems as Physical Data Processors: Evolution as a Case of Embodied Computing
The prevailing view of biological evolution is not unlike bricolage/pastiche/tinkering—in sharp contrast with rational engineering. Yet, different paths often lead to solutions that coincide or converge whether they emerge from naturally...
04
DOI 10.1002/bies.70192
Open DOI
Interplay Between Motor Proteins and Microtubule Stability: Involvement of Tubulin PTMs and MAPs
Microtubules and motor proteins are components of a cytoskeleton that influence each other to maintain cellular functions. Microtubules act as tracks for motor protein‐driven intracellular transport of organelles, vesicles, and macromole...
05
DOI 10.1002/bies.70193
Open DOI
Metabolic Structure: A Paradigm Revisited in Light of the Fundamental Parameters of Living Systems
The concept of ‘metabolic structure’, which considers metabolism as a stable, genetically encoded architecture with enzymes occupying fixed control positions, is indispensable but incomplete, and it obscures metabolism's true nature. I p...
06
DOI 10.1002/bies.70176
Open DOI
Correction to “Rethinking One Health: Microbial Foundations for Ecological Governance”
07
DOI 10.1002/bies.70195
Open DOI
Protein‐Ratio Rheostats: Integrating Stoichiometry, Abundance, and Binding Affinity in Cellular Function
Cellular function is frequently interpreted in terms of gene presence, protein abundance, and dose–response relationships. Yet interacting proteins operate within binding and assembly regimes in which relative abundance can alter which m...
08
DOI 10.1002/bies.70185
Open DOI
Molecular Control of Replication Fork Speed: dNTP Availability, Phosphorylation, PARylation, and UFMylation
The regulation of replication fork (RF) speed is essential for faithful genome duplication and the maintenance of genome stability, and its dysregulation contributes to carcinogenesis and developmental disorders. Although many anticancer...
09
DOI 10.1002/bies.70190
Open DOI
Controlling Meiotic Double‐Strand Break Formation in Mice: A Web of Multivalent Protein–Protein Interactions
A complex network of protein interactions promotes the cooperative assembly of biomolecular condensates that control the formation of meiotic DNA double‐strand breaks (DSBs). In many species, programmed DSBs made by SPO11 are required fo...
10
DOI 10.1002/bies.70187
Open DOI
Kinetic Control of Condensate Function: How the Formation Dynamics of APBs Influence ALT Cancer Telomere Length Heterogeneity
Telomerase‐free cancer cells use a recombination‐based alternative lengthening of telomeres (ALT) pathway to maintain telomere length. ALT cancer cells exhibit extreme heterogeneity in telomere length, which requires the formation of a s...
Bibliographical & Catalog Notes
Print issues ceased with v. 36 (2014).
Issue for July 1984 called also "First issue."
Vol. 8, no. 3 (1988) published as a combined issue with v. 8, no. 2.
Also issued online.
Cosponsored by various members of the International Council of Scientific Unions
Vol. 10, no. 1 (1989)19920824 Notice not published Printed on acid-free paper