Impact Factor
4.31
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
6.54
2,333 total citations
h5-index
53
h5 depth signal
Cited share
90.3%
346 of 383
Total citations
2,333
Citation footprint
Metric signal
6.43
External metric anchor
Self-citation
0.3%
8 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
United Kingdom
584
United States
496
Australia
168
Germany
130
China
80
Citing Fields of Research
Cell Biology
610
Genetics & Genomics
365
Cellular Membrane Transporters & Receptors
189
Cancer, Autophagy & Apoptosis
181
Immunology
157
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
30,735
Articles tracked across the journal lifetime
Indexed publication set
383
346 cited publications
Last 12 months
126
10.5 publications/month
Open access share
99.2%
125 OA publications in the last year
Leading citing country
United Kingdom
584 citing publications
Leading citing field
Cell Biology
610 citing publications
Latest tracked publication
13 October 2026
10 recent records available
Coverage & Indexing History
v1n4, Jul. 1973-
v1n4, Jul. 1973-
Active Coverage
Research Focus & Classification
Indexed Subjects
Cell Biology
Genetics & Genomics
Bacteriology
Cancer, Autophagy & Apoptosis
Immunology
DNA Damage
Issuing & Sponsoring Bodies
Contributor
Biochemical Society (Great Britain)
Broad Categories
Biochemistry
Cell Biology
Genetics & Genomics
DNA Damage
Cancer, Autophagy & Apoptosis
Mitochondria
Cancer & Development
Neurodegenerative Disorders
MeSH Descriptors
Biochemistry
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
DOI 10.1042/bst20260632
Open DOI
Increasing autophagy with fasting and diet: separating fact from fiction
Autophagy is the cell’s recycling system; it removes unwanted or dysfunctional cellular material, thereby preventing damage. Importantly, it is a key pro-survival pathway during periods of stress, for instance, during nutrient scarcity. ...
02
DOI 10.1042/bst20260842
Open DOI
NINJ1-mediated membrane rupture bridges cell death to tissue inflammation
Plasma membrane rupture (PMR) is a terminal event in many forms of lytic cell death, yet was long regarded as a passive consequence of osmotic swelling and mechanical membrane failure. The identification of ninjurin-1 (NINJ1) as a key me...
03
DOI 10.1042/bst20250199
Open DOI
How do low-complexity domain interactions control transcription?
A substantial portion of the proteome consists of intrinsically disordered regions (IDRs) that do not fold into well-defined structures. IDRs often comprise low-complexity domains (LCDs), whose repetitive, compositionally biased sequence...
04
DOI 10.1042/bst20250531
Open DOI
Pathway(s) and distribution of photosystem II-cyclic electron flow
Photosystem II is a highly conserved protein complex found across all oxygenic phototrophs and the source of almost all biologically available electrons and atmospheric oxygen. This large, membrane-bound complex is at continuous risk of ...
05
DOI 10.1042/bst20250382
Open DOI
p38γ and p38δ MAP kinases in inflammation
The p38γ and p38δ mitogen-activated protein kinases (MAPKs) are members of the p38 family that have emerged as important regulators of inflammatory signaling. Initially considered minor or alternative isoforms, accumulating evidence has ...
06
DOI 10.1042/bst20250546
Open DOI
Genes, fuel, and fate in lung cancer
Cellular plasticity is a central feature of malignant progression, allowing tumour cells to change identity during tumour evolution, therapeutic adaptation, and metastatic dissemination. This issue is especially acute in lung cancer, whe...
07
DOI 10.1042/bst20250120
Open DOI
Manipulation of the cell cycle by HIV-1
Many viruses manipulate the host cell cycle to enhance viral replication and promote their evasion of host immune defenses. Human immunodeficiency virus type 1 (HIV-1) is no exception and triggers cell cycle arrest during its infection o...
08
DOI 10.1042/bst20260203
Open DOI
Recent advances in understanding the regulatory mechanisms of P4-ATPase lipid flippases
P4-ATPases establish and maintain transbilayer lipid asymmetry in eukaryotic membranes by transporting phospholipids from the exoplasmic to the cytosolic membrane leaflet at the expense of ATP hydrolysis. In recent years, biochemical ana...
09
DOI 10.1042/bst20260103
Open DOI
Folate transporters at the plasma membrane of fungi and plants: specificity, mechanism of transport, and importance
Folates are essential metabolites of the cell and need to be acquired either by transport or biosynthesis. All metazoa including humans, some bacteria, and some protozoa acquire it solely through specific transporters. Plants and fungi, ...
10
DOI 10.1042/bst20260704
Open DOI
One adaptor, multiple outcomes: decoding TRIFosome heterogeneity
Innate immune signalling is uniquely driven through the formation of supramolecular organising centres (SMOCs), which couple receptor activation with downstream effector recruitment. The adaptor protein TRIF coordinates interferon, infla...
Bibliographical & Catalog Notes
Also issued online.
Vol. 11, no. 1 (1983)19920824 Notice not published Printed on acid-free paper