Impact Factor
4.42
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
7.69
1,887 total citations
h5-index
62
h5 depth signal
Cited share
91.0%
232 of 255
Total citations
1,887
Citation footprint
Metric signal
8.02
External metric anchor
Self-citation
1.1%
20 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
United States
517
China
432
Germany
125
Italy
120
United Kingdom
98
Citing Fields of Research
Molecular Pharmacology
533
Cancer, Autophagy & Apoptosis
219
Mitochondria
199
Immunology
171
Neurodegenerative Disorders
129
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
5,066
Articles tracked across the journal lifetime
Indexed publication set
255
232 cited publications
Last 12 months
119
9.9 publications/month
Open access share
3.4%
4 OA publications in the last year
Leading citing country
United States
517 citing publications
Leading citing field
Molecular Pharmacology
533 citing publications
Latest tracked publication
7 October 2026
10 recent records available
Coverage & Indexing History
v1n1, spring 1999-
v1n1, spring 1999-
Active Coverage
Research Focus & Classification
Indexed Subjects
Cancer, Autophagy & Apoptosis
Molecular Pharmacology
Mitochondria
Neurodegenerative Disorders
Immunology
Kidney Diseases
Issuing & Sponsoring Bodies
Contributor
Mary Ann Liebert, Inc
Broad Categories
Metabolism
Molecular Pharmacology
Cancer, Autophagy & Apoptosis
Mitochondria
Aging & Longevity
MeSH Descriptors
Antioxidants
Oxidation-Reduction
Signal Transduction
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
DOI 10.1177/15230864261494911
Open DOI
Identification of Thioredoxin-Interacting Protein as an Upstream Regulator of Ferroptosis in Hyperuricemic Nephropathy
Aim: Hyperuricemic nephropathy (HN) is a major contributor to chronic kidney disease, yet current urate-lowering therapies often fail to halt tubular injury once established. While ferroptosis is known to drive renal injury in HN, the up...
02
DOI 10.1177/15230864261495029
Open DOI
A SIRT1-PERK-HSPA5-MARCHF1 Proteostasis Axis Controls GPX4 Stability and Ferroptotic Susceptibility in Hepatic Stellate Cells
Aims: Liver fibrosis is a chronic inflammatory condition characterized by excessive extracellular matrix accumulation driven by hepatic stellate cell (HSC) activation, and inducing HSC ferroptosis represents an effective strategy to rest...
03
DOI 10.1177/15230864261494912
Open DOI
Loss of Selenot in POMC Neurons Disrupts ER Homeostasis and Induces A Senescent-like Phenotype
Aims: Selenoprotein T (SELENOT) is an endoplasmic reticulum (ER)-resident thioredoxin-like oxidoreductase implicated in redox and calcium (Ca 2+ ) homeostasis and protein quality control. Although SELENOT is highly expressed in the brain...
04
DOI 10.1177/15230864261490413
Open DOI
Metabolite Regulation of Mitochondrial Quality Control and Regulated Cell Death
Background: Altered metabolism contributes to disease progression and variable treatment response, but metabolite abundance alone rarely determines whether stressed cells recover or undergo regulated cell death (RCD). Scope of Review: Th...
05
DOI 10.1177/15230864261489976
Open DOI
A Unified Mechanistic Framework for Copper Toxicity in Neutrophils: Metabolic Rewiring, Cell-Death Crosstalk, and Immune Dysfunction
Significance: Copper is an essential redox-active micronutrient, but failure of copper compartmentalization can convert it from a catalytic cofactor into a source of oxidative, proteotoxic, and mitochondrial stress. Neutrophils are a pla...
06
07
DOI 10.1177/15230864261489484
Open DOI
Beyond Cys65: Redox Effector Factor-1 CRISPR-Engineered Cys93 and Cys99 Mutants Reveal Cooperative and Secondary Roles of Redox Signaling and Pancreatic Ductal Adenocarcinoma Progression
Aims: Apurinic/apyrimidinic endonuclease 1/redox effector factor 1 (APE1/Ref-1) is a multifunctional protein implicated in pancreatic ductal adenocarcinoma (PDAC) through its redox regulation of oncogenic transcription factors (TFs) such...
08
DOI 10.1177/15230864261486899
Open DOI
Hypoxic Preconditioning Prevents the Neonatal Lung Against Hyperoxia-Induced Ferroptosis Through the Nrf2–CBS Axis
Aims: Hyperoxia-induced oxidative injury contributes to ferroptosis and abnormal lung remodeling in preterm infants receiving oxygen therapy. We previously demonstrated that postnatal hypoxic preconditioning (HP) reduces hyperoxia-induce...
09
DOI 10.1177/15230864261485730
Open DOI
Retraction: “Lipopolysaccharide-Induced Mitochondrial DNA Depletion”
10
DOI 10.1177/15230864261487908
Open DOI
SGK2 Promotes NEDD4L-Mediated ACSL4 Ubiquitination to Inhibit Ferroptosis and Alleviate Diabetic Kidney Disease
Aims: Despite major advances with sodium–glucose cotransporter 2 (SGLT2) inhibitors and glucagon-like peptide-1 (GLP-1) receptor agonists, progressive tubulointerstitial injury remains a major determinant of renal function decline in dia...
Bibliographical & Catalog Notes
Title from cover.
Also issued online.
Vol.1, no. 3 (Dec 1999)20001117 Notice published Printed on acid-free paper that meets the full ANSI standard for Permanent Paper