Impact Factor
3.75
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
3.69
1,133 total citations
h5-index
38
h5 depth signal
Cited share
77.7%
255 of 328
Total citations
1,133
Citation footprint
Metric signal
4.35
External metric anchor
Self-citation
6.9%
78 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
United States
1,497
China
249
Japan
140
Germany
128
Denmark
92
Citing Fields of Research
Kidney Diseases
1,482
Physiology
629
Urology
474
Cell Biology
235
Cellular Membrane Transporters & Receptors
213
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
13,119
Articles tracked across the journal lifetime
Indexed publication set
328
255 cited publications
Last 12 months
162
13.5 publications/month
Open access share
17.9%
29 OA publications in the last year
Leading citing country
United States
1,497 citing publications
Leading citing field
Kidney Diseases
1,482 citing publications
Latest tracked publication
1 November 2026
10 recent records available
Coverage & Indexing History
v278n1, Jan. 2000-
v278n1, Jan. 2000-
Active Coverage
Research Focus & Classification
Indexed Subjects
Kidney Diseases
Physiology
Urology
Diabetes
Issuing & Sponsoring Bodies
Contributor
American Physiological Society (1887- )
Broad Categories
Nephrology
Physiology
Urology
Cancer & Development
Diabetes
MeSH Descriptors
Kidney
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
DOI 10.1152/ajprenal.00376.2025_cor
Open DOI
Correction for Budnick et al., volume 330, 2026, p. F683–F701
02
DOI 10.1152/ajprenal.00170.2012_ret
Open DOI
Retraction for Salhan et al., volume 303, 2012, p. F503–F514
03
DOI 10.1152/ajprenal.00160.2026
Open DOI
Glucagon receptor agonist aggravates a murine model of Balkan nephropathy
This study suggests that a glucagon receptor agonist (GCGRA) can aggravate a murine model of Balkan nephropathy induced by the nephrotoxin aristolochic acid. Mechanistically, this is associated with GCGRA-induced upregulation of the inhi...
04
DOI 10.1152/ajprenal.00033.2026
Open DOI
Moderate loss of renal mass exacerbates renal damage in a genetic mouse model of high-renin hypertension
A new mouse model for development of a strong chronic kidney disease (CKD) phenotype without the need for highly burdening interventions has been established. Genetically caused high-renin hypertension leads to typical signs of CKD such ...
05
DOI 10.1152/ajprenal.90084.2026
Open DOI
CRISPR activation of endogenous PKD1 increases polycystin-1 levels and suppresses cellular features of ADPKD
This study provides proof-of-concept for a CRISPR activation (CRISPRa)-based approach to ADPKD. CRISPRa targeting the PKD1 promoter increased polycystin-1 levels in mouse and human cells. Upregulation of a hypomorphic Pkd1 allele increas...
06
DOI 10.1152/ajprenal.00629.2009_ret
Open DOI
Retraction for Mikulak et al., volume 299, 2010, p. F664–F673
07
DOI 10.1152/ajprenal.00024.2011_ret
Open DOI
Retraction for Salhan et al., volume 302, 2012, p. F129–F140
08
DOI 10.1152/ajprenal.00194.2026
Open DOI
A model of recurrent heat stress that causes kidney injury in mice
Here we present a recurrent heat stress model that produces consistent increases in core temperature and dehydration with each event, leading to kidney injury in male and female mice. This robust and reproducible model demonstrates that ...
09
DOI 10.1152/ajprenal.00189.2026
Open DOI
Differential regulation of basal and interferon-induced APOL1 gene expression in podocytes
APOL1 kidney disease is triggered by events that induce high levels of APOL1 expression. We show here that basal and induced APOL1 expression are regulated by different mechanisms, with induced expression largely dependent on interferon ...
10
DOI 10.1152/ajprenal.00096.2026
Open DOI
Impact of renal iron overload on sickle cell nephropathy in a humanized mouse model
SCD, an inherited hemolytic anemia, is associated with increased mortality from prolonged iron overload. Current interventional strategies are limited to the chelation of transfusion-mediated iron overload only, leaving kidney-specific, ...
Bibliographical & Catalog Notes
Title from cover.
Print issues ceased with v. 301, no. 6 (2011).
Vols. for <2000-> also carry the numbering of the consolidated version of American journal of physiology; i.e., v. <47-> also numbered as v. <278-> .
Also issued online.
Also issued as a section of the consolidated version of The American journal of physiology.