Impact Factor
4.56
Journal Citation Impact
Estimated influence, citation density, publishing volume, and citation integrity from the analytics table.
Citation rate
8.43
3,515 total citations
h5-index
70
h5 depth signal
Cited share
90.9%
400 of 440
Total citations
3,515
Citation footprint
Metric signal
7.23
External metric anchor
Self-citation
3.3%
117 self-citations
Citations Trend
Monthly Publications Cadence
Citing Geographic Demographics
China
948
United States
386
Spain
67
Germany
63
Italy
60
Citing Fields of Research
Neurodegenerative Disorders
509
Aging & Longevity
449
Immunology
128
Musculoskeletal Disorders
118
DNA Damage
96
Publishing Activity & Reach
Volume, recency, access pattern, and citing-community signals from the journal analytics record.
All-time article footprint
1,783
Articles tracked across the journal lifetime
Indexed publication set
440
400 cited publications
Last 12 months
161
161.0 publications/month
Open access share
49.7%
80 OA publications in the last year
Leading citing country
China
948 citing publications
Leading citing field
Neurodegenerative Disorders
509 citing publications
Latest tracked publication
1 January 2026
10 recent records available
Coverage & Indexing History
v14n1, Jan. 2023-
v1, 2010-
Active Coverage
Research Focus & Classification
Indexed Subjects
Aging & Longevity
Neurodegenerative Disorders
Issuing & Sponsoring Bodies
Contributor
Buck Institute for Age Research
Broad Categories
Geriatrics
Neurodegenerative Disorders
DNA Damage
Aging & Longevity
Cancer, Autophagy & Apoptosis
Musculoskeletal Disorders
Mitochondria
MeSH Descriptors
Aging
Latest Publications
Recent article records with DOI-first linking and condensed abstracts.
01
DOI 10.14336/ad.2026.1096
Open DOI
Exercise, Dynamic Functional Connectivity, and Sex in Alzheimer’s Disease: A Hypothesis-Generating Framework for the Sex-Dependent Dynamic Reconstitution Model
Alzheimer’s disease (AD) is increasingly viewed as a disorder of large-scale brain network dynamics. Whether exercise-related changes in dynamic functional connectivity (dFC) reflect neural reorganization, vascular effects, or both remai...
02
DOI 10.14336/ad.2026.0844
Open DOI
Aging Intestinal Niche: Regulation of Stem Cells and Disease
The maintenance of intestinal epithelial homeostasis and regenerative potential is fundamentally orchestrated by intestinal stem cells (ISCs). Aging is associated with a pronounced decline in intestinal function, with ISC dysfunction eme...
03
DOI 10.14336/ad.2026.1114
Open DOI
Biomarker-Guided, Stage-Specific Senotherapy for Metabolic Dysfunction-Associated Steatotic Liver Disease
Cellular senescence drives inflammation and fibrosis progression in metabolic dysfunction-associated steatotic liver disease (MASLD) through the senescence-associated secretory phenotype (SASP). Existing research covers hepatocyte types,...
04
DOI 10.14336/ad.2026.1040
Open DOI
A Clearance-Repair-Activation Framework for Alzheimer's Disease: A Perspective and Validation Roadmap
Alzheimer's disease (AD) is a heterogeneous clinicobiological syndrome involving impaired protein clearance, neuroinflammation, synaptic and cellular injury, network dysfunction, and progressive functional decline. Amyloid-directed antib...
05
DOI 10.14336/ad.2026.0896
Open DOI
Aging as a Potential Modulator of Extramedullary Hematopoiesis: A Hypothesis-Generating Niche-HSPC Competence-Trigger Model
Extramedullary hematopoiesis (EMH) is a compensatory response to hematopoietic stress that occurs unevenly across disorders with ineffective erythropoiesis (IE). The mechanisms underlying differing EMH patterns despite similarly severe I...
06
DOI 10.14336/ad.2026.0888
Open DOI
Neuroimmune State Configurations at the Amyloid-Tau Interface in Alzheimer’s Disease: A Testable Framework
Neuroimmune mechanisms are central to Alzheimer’s disease (AD), but a single continuum of “more inflammation” does not explain why similar amyloid-β (Aβ) and tau burdens are associated with different clinical trajectories, tissue respons...
07
DOI 10.14336/ad.2026.0886
Open DOI
Sympathetic Regulation of Bone Metabolism: Mechanisms across Homeostasis, Aging and Disease
Bone remodeling is regulated by endocrine, mechanical, paracrine, immune, vascular, and neural signals. This narrative review examines sympathetic regulation of bone metabolism within an asymmetric bidirectional brain-bone neural network...
08
DOI 10.14336/ad.2026.0869
Open DOI
Senescence and SASP in the Stromal Microenvironment of Breast Cancer: Dual Role in Progression and Therapeutic Options
Cellular senescence, a process characterized by the typically persistent cell-cycle arrest that occurs in response to DNA damage, plays a dual role in carcinogenesis. This complexity is predominantly influenced by the presence of senesce...
09
DOI 10.14336/ad.2026.0702
Open DOI
GDF15 Induces Weight Loss with Diet- and Age-Dependent Metabolic Effects Across Tissues in Mice
Growth differentiation factor 15 (GDF15) is a regulator of body weight and systemic homeostasis. However, the mechanisms underlying its effects remain unclear. This study aimed to determine whether diet, age, and sex influence GDF15’s me...
10
DOI 10.14336/ad.2026.0679
Open DOI
Mitochondria Beyond Bioenergetics: Canonical and Non-Canonical Functions in Ischemic Stroke and Aging
Ischemic stroke remains a leading cause of death and long-term disability worldwide, yet effective neuroprotective therapies beyond reperfusion strategies are still lacking. Emerging evidence highlights mitochondria as central regulators...